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Dr Yasir Ahmed Syed
- Ar gael fel goruchwyliwr ôl-raddedig
Timau a rolau for Yasir Ahmed Syed
Uwch Ddarlithydd
Niwrowyddoniaeth
Trosolwg
Rwy'n niwrowyddonydd sy'n arbenigo yn y mecanweithiau biolegol sy'n sail i anhwylderau niwro-ddatblygiadol a niwroseiciatrig, gan gynnwys anhwylder sbectrwm awtistiaeth (ASD), anabledd deallusol, sgitsoffrenia, ac anhwylderau hwyliau. Mae fy ymchwil yn canolbwyntio ar ddeall sut mae ffactorau risg genetig yn tarfu ar ddatblygiad a swyddogaeth yr ymennydd, gyda'r nod o nodi targedau therapiwtig newydd.
Yn fy labordy, rydym yn defnyddio bôn-gelloedd pluripotent sy'n deillio o gleifion (iPSCs) i fodelu datblygiad ymennydd dynol in vitro. Trwy wahaniaethu'r celloedd hyn yn llinellau niwral ac organoidau ymennydd 3D, rydym yn ymchwilio i fecanweithiau clefyd ar y lefel gellog a moleciwlaidd.
Mae ein gwaith yn cael ei yrru gan ddull amlddisgyblaethol, gan integreiddio technegau cellog, genetig, electroffisiolegol, ymddygiadol a gwyddor deunyddiau i astudio cychwyn a dilyniant clefydau. Yn y pen draw, ein nod yw datgelu targedau cyffuriau dibynadwy a all lywio strategaethau therapiwtig yn y dyfodol ar gyfer anhwylderau cymhleth yr ymennydd.
Cyhoeddiad
2026
- Singh, T. et al., 2026. Human induced pluripotent stem cell-derived microglia with 1q21.1 deletion and duplication exhibit aberrant inflammatory response. Genes & diseases 13 (4) 101923. (10.1016/j.gendis.2025.101923)
- Thahani, I. et al., 2026. The effectiveness of teletherapy for developmental language disorder in India. Frontiers in Digital Health 8 1827565. (10.3389/fdgth.2026.1827565)
- Durmaz, E. et al., 2026. Extracellular vesicle–mediated delivery of miR-181a-3p confers neuroprotection to degenerating retinal ganglion cells. Experimental Eye Research 265 110882. (10.1016/j.exer.2026.110882)
- Casella, C. et al. 2026. Differences in white matter detected by ex vivo 9.4 T MRI are associated with axonal changes in the R6/1 model of Huntington's disease. Neurobiology of Disease 220 107318. (10.1016/j.nbd.2026.107318)
- Wang, T. et al. 2026. How copy number variations shape brain developmental disorders: unravelling the synaptic puzzle. Psychiatry and Clinical Neurosciences 80 (3), pp.166-179. (10.1111/pcn.70009)
2025
- Kidson, C. , Loh, N. and Syed, Y. A. 2025. Mortality in tuberous sclerosis complex in the United Kingdom 2016-2022. Journal of Intellectual Disability Research 69 (6), pp.457-464. (10.1111/jir.13225)
- Mikaeloff, F. et al., 2025. Host plasma microenvironment in immunometabolically impaired HIV infection leads to dysregulated monocyte function and synaptic transmission ex vivo. Advanced Science 12 (16) 2416453. (10.1002/advs.202416453)
- Rao, P. et al., 2025. Neuroprotective approach, microbiome therapy, and overcoming insulin resistance in the brain. In: Ashraf, G. M. et al., Multi-Factorial Approach as a Therapeutic Strategy for the Management of Alzheimer’s Disease. Singapore: Springer. , pp.259-280. (10.1007/978-981-96-0259-9_13)
- Wang, T. et al. 2025. Mice with 16p11.2 deletion and duplication show alterations in biological processes associated with white matter. International Journal of Molecular Sciences 26 (2) 573. (10.3390/ijms26020573)
2024
- Mitchell, M. , Spasova, A. and Syed, Y. A. 2024. Unraveling autism: using brain organoids to investigate sex differences in brain development. Biological Psychiatry 4 (5) 100360. (10.1016/j.bpsgos.2024.100360)
- Stojanovska, I. et al., 2024. The utilization of psychopharmacological treatments for individuals with autism spectrum disorder (ASD) in a middle-income European country. Research in Autism Spectrum Disorders 111 102329. (10.1016/j.rasd.2024.102329)
- Jaddoh, A. et al. 2024. Interacting with smart virtual assistants for individuals with dysarthria: a comparative study on usability and user preferences. Applied Sciences 14 (4) 1409. (10.3390/app14041409)
2023
- Alam, A. et al., 2023. Modelling the inflammatory response of traumatic brain injury using human induced pluripotent stem cell derived microglia. Journal of Neurotrauma 40 (19-20), pp.2164-2173. (10.1089/neu.2022.0508)
- Nicolaou, K. et al., 2023. Game based learning rehabilitation for children with speech disabilities: Presenting two bespoke video games. Presented at: BCS HCI: 36th International BCS Human-Computer Interaction Conference York, UK 28-29 August 2023.
- Mohammad, F. et al., 2023. Editorial: Advances in understanding synaptic function and its dysfunction in neurological disorders. Frontiers in Molecular Neuroscience 16 (10.3389/fnmol.2023.1239315)
- Koceski, A. et al., 2023. Understanding the relationship between distress behaviour and health status of people with autism spectrum disorder. Healthcare 11 (11) 1565. (10.3390/healthcare11111565)
- Smith, C. J. et al., 2023. Unravelling the clinical co-morbidity and risk factors associated with agenesis of the corpus callosum. Journal of Clinical Medicine 12 (11) 3623. (10.3390/jcm12113623)
2022
- Robinson, J. et al., 2022. The association of neurodevelopmental abnormalities, congenital heart and renal defects in a Tuberous Sclerosis Complex patient cohort. BMC Medicine 20 123. (10.1186/s12916-022-02325-0)
- Dash, S. , Syed, Y. A. and Khan, M. R. 2022. Understanding the role of the gut microbiome in brain development and its association with neurodevelopmental psychiatric disorders. Frontiers in Cell and Developmental Biology 10 880544. (10.3389/fcell.2022.880544)
- Chapman, G. et al. 2022. Using induced pluripotent stem cells to investigate human neuronal phenotypes in 1q21.1 deletion and duplication syndrome. Molecular Psychiatry 27 , pp.819-830. (10.1038/s41380-021-01182-2)
2021
- Dowden, L. et al. 2021. Contribution of congenital heart disorders associated with copy number variants in mediating risk for brain developmental disorders; evidence from 20-year retrospective cohort study. Frontiers in Cardiovascular Medicine 8 655463. (10.3389/fcvm.2021.655463)
2020
- Drakulic, D. et al., 2020. Copy number variants (CNVs): a powerful tool for iPSC-based modelling of ASD. Molecular Autism 11 (1) 42. (10.1186/s13229-020-00343-4)
- Akter, F. et al., 2020. The pathophysiology of degenerative cervical myelopathy and the physiology of recovery following decompression. Frontiers in Neuroscience 14 138. (10.3389/fnins.2020.00138)
- Savory, K. et al. 2020. Impact of copy number variation on human neurocognitive deficits and congenital heart defects: a systematic review. Neuroscience and Biobehavioral Reviews 108 , pp.83-93. (10.1016/j.neubiorev.2019.10.020)
- Savory, K. and Syed, Y. A. 2020. Advances in the understanding of cellular pathogenesis associated with Autism Spectrum Disorder. Journal for ReAttach Therapy and Developmental Diversities 2 (2), pp.96-118.
2019
- Silva, A. I. et al., 2019. Cyfip1 haploinsufficient rats show white matter changes, myelin thinning, abnormal oligodendrocytes and behavioural inflexibility. Nature Communications 10 3455. (10.1038/s41467-019-11119-7)
2017
- Radtke, F. et al. 2017. Modulating neuroinflammation to treat neuropsychiatric disorders. BioMed Research International 5071786.
2016
- Syed, Y. A. , Abdulla, S. A. and Kotter, M. R. N. 2016. Studying the effects of semaphorins on oligodendrocyte lineage cells. In: Terman, J. R. ed. Semaphorin Signaling. Vol. 1493, Methods in Molecular Biology New York, NY: Humana Press. , pp.363-378. (10.1007/978-1-4939-6448-2_26)
- Gonzalez, G. A. et al., 2016. Tamoxifen accelerates the repair of demyelinated lesions in the central nervous system. Scientific Reports 6 31599. (10.1038/srep31599)
- Dhillon, R. S. et al., 2016. Axonal plasticity underpins the functional recovery following surgical decompression in a rat model of cervical spondylotic myelopathy. Acta Neuropathologica Communications 4 89. (10.1186/s40478-016-0359-7)
- Syed, Y. A. et al. 2016. Antibody-mediated neutralization of myelin-associated EphrinB3 accelerates CNS remyelination. Acta Neuropathologica 131 (2), pp.281-298. (10.1007/s00401-015-1521-1)
2013
- Syed, Y. A. et al. 2013. Inhibition of phosphodiesterase-4 promotes oligodendrocyte precursor cell differentiation and enhances CNS remyelination. EMBO Molecular Medicine 5 (12), pp.1918-1934. (10.1002/emmm.201303123)
- Hannan, N. et al., 2013. Generation of multipotent foregut stem cells from human pluripotent stem cells. Stem Cell Reports 1 (4), pp.293-306. (10.1016/j.stemcr.2013.09.003)
2011
- Syed, Y. A. et al. 2011. Inhibition of CNS remyelination by the presence of semaphorin 3A. Journal of Neuroscience 31 (10), pp.3719-3728. (10.1523/JNEUROSCI.4930-10.2011)
2009
- Baer, A. S. et al., 2009. Myelin-mediated inhibition of oligodendrocyte precursor differentiation can be overcome by pharmacological modulation of Fyn-RhoA and protein kinase C signalling. Brain 132 (2), pp.465-481. (10.1093/brain/awn334)
2008
- Syed, Y. A. et al. 2008. Inhibition of oligodendrocyte precursor cell differentiation by myelin-associated proteins. Neurosurgical Focus 24 (3-4) E5. (10.3171/FOC/2008/24/3-4/E4)
Adrannau llyfrau
- Rao, P. et al., 2025. Neuroprotective approach, microbiome therapy, and overcoming insulin resistance in the brain. In: Ashraf, G. M. et al., Multi-Factorial Approach as a Therapeutic Strategy for the Management of Alzheimer’s Disease. Singapore: Springer. , pp.259-280. (10.1007/978-981-96-0259-9_13)
- Syed, Y. A. , Abdulla, S. A. and Kotter, M. R. N. 2016. Studying the effects of semaphorins on oligodendrocyte lineage cells. In: Terman, J. R. ed. Semaphorin Signaling. Vol. 1493, Methods in Molecular Biology New York, NY: Humana Press. , pp.363-378. (10.1007/978-1-4939-6448-2_26)
Cynadleddau
- Nicolaou, K. et al., 2023. Game based learning rehabilitation for children with speech disabilities: Presenting two bespoke video games. Presented at: BCS HCI: 36th International BCS Human-Computer Interaction Conference York, UK 28-29 August 2023.
Erthyglau
- Singh, T. et al., 2026. Human induced pluripotent stem cell-derived microglia with 1q21.1 deletion and duplication exhibit aberrant inflammatory response. Genes & diseases 13 (4) 101923. (10.1016/j.gendis.2025.101923)
- Thahani, I. et al., 2026. The effectiveness of teletherapy for developmental language disorder in India. Frontiers in Digital Health 8 1827565. (10.3389/fdgth.2026.1827565)
- Durmaz, E. et al., 2026. Extracellular vesicle–mediated delivery of miR-181a-3p confers neuroprotection to degenerating retinal ganglion cells. Experimental Eye Research 265 110882. (10.1016/j.exer.2026.110882)
- Casella, C. et al. 2026. Differences in white matter detected by ex vivo 9.4 T MRI are associated with axonal changes in the R6/1 model of Huntington's disease. Neurobiology of Disease 220 107318. (10.1016/j.nbd.2026.107318)
- Wang, T. et al. 2026. How copy number variations shape brain developmental disorders: unravelling the synaptic puzzle. Psychiatry and Clinical Neurosciences 80 (3), pp.166-179. (10.1111/pcn.70009)
- Kidson, C. , Loh, N. and Syed, Y. A. 2025. Mortality in tuberous sclerosis complex in the United Kingdom 2016-2022. Journal of Intellectual Disability Research 69 (6), pp.457-464. (10.1111/jir.13225)
- Mikaeloff, F. et al., 2025. Host plasma microenvironment in immunometabolically impaired HIV infection leads to dysregulated monocyte function and synaptic transmission ex vivo. Advanced Science 12 (16) 2416453. (10.1002/advs.202416453)
- Wang, T. et al. 2025. Mice with 16p11.2 deletion and duplication show alterations in biological processes associated with white matter. International Journal of Molecular Sciences 26 (2) 573. (10.3390/ijms26020573)
- Mitchell, M. , Spasova, A. and Syed, Y. A. 2024. Unraveling autism: using brain organoids to investigate sex differences in brain development. Biological Psychiatry 4 (5) 100360. (10.1016/j.bpsgos.2024.100360)
- Stojanovska, I. et al., 2024. The utilization of psychopharmacological treatments for individuals with autism spectrum disorder (ASD) in a middle-income European country. Research in Autism Spectrum Disorders 111 102329. (10.1016/j.rasd.2024.102329)
- Jaddoh, A. et al. 2024. Interacting with smart virtual assistants for individuals with dysarthria: a comparative study on usability and user preferences. Applied Sciences 14 (4) 1409. (10.3390/app14041409)
- Alam, A. et al., 2023. Modelling the inflammatory response of traumatic brain injury using human induced pluripotent stem cell derived microglia. Journal of Neurotrauma 40 (19-20), pp.2164-2173. (10.1089/neu.2022.0508)
- Mohammad, F. et al., 2023. Editorial: Advances in understanding synaptic function and its dysfunction in neurological disorders. Frontiers in Molecular Neuroscience 16 (10.3389/fnmol.2023.1239315)
- Koceski, A. et al., 2023. Understanding the relationship between distress behaviour and health status of people with autism spectrum disorder. Healthcare 11 (11) 1565. (10.3390/healthcare11111565)
- Smith, C. J. et al., 2023. Unravelling the clinical co-morbidity and risk factors associated with agenesis of the corpus callosum. Journal of Clinical Medicine 12 (11) 3623. (10.3390/jcm12113623)
- Robinson, J. et al., 2022. The association of neurodevelopmental abnormalities, congenital heart and renal defects in a Tuberous Sclerosis Complex patient cohort. BMC Medicine 20 123. (10.1186/s12916-022-02325-0)
- Dash, S. , Syed, Y. A. and Khan, M. R. 2022. Understanding the role of the gut microbiome in brain development and its association with neurodevelopmental psychiatric disorders. Frontiers in Cell and Developmental Biology 10 880544. (10.3389/fcell.2022.880544)
- Chapman, G. et al. 2022. Using induced pluripotent stem cells to investigate human neuronal phenotypes in 1q21.1 deletion and duplication syndrome. Molecular Psychiatry 27 , pp.819-830. (10.1038/s41380-021-01182-2)
- Dowden, L. et al. 2021. Contribution of congenital heart disorders associated with copy number variants in mediating risk for brain developmental disorders; evidence from 20-year retrospective cohort study. Frontiers in Cardiovascular Medicine 8 655463. (10.3389/fcvm.2021.655463)
- Drakulic, D. et al., 2020. Copy number variants (CNVs): a powerful tool for iPSC-based modelling of ASD. Molecular Autism 11 (1) 42. (10.1186/s13229-020-00343-4)
- Akter, F. et al., 2020. The pathophysiology of degenerative cervical myelopathy and the physiology of recovery following decompression. Frontiers in Neuroscience 14 138. (10.3389/fnins.2020.00138)
- Savory, K. et al. 2020. Impact of copy number variation on human neurocognitive deficits and congenital heart defects: a systematic review. Neuroscience and Biobehavioral Reviews 108 , pp.83-93. (10.1016/j.neubiorev.2019.10.020)
- Savory, K. and Syed, Y. A. 2020. Advances in the understanding of cellular pathogenesis associated with Autism Spectrum Disorder. Journal for ReAttach Therapy and Developmental Diversities 2 (2), pp.96-118.
- Silva, A. I. et al., 2019. Cyfip1 haploinsufficient rats show white matter changes, myelin thinning, abnormal oligodendrocytes and behavioural inflexibility. Nature Communications 10 3455. (10.1038/s41467-019-11119-7)
- Radtke, F. et al. 2017. Modulating neuroinflammation to treat neuropsychiatric disorders. BioMed Research International 5071786.
- Gonzalez, G. A. et al., 2016. Tamoxifen accelerates the repair of demyelinated lesions in the central nervous system. Scientific Reports 6 31599. (10.1038/srep31599)
- Dhillon, R. S. et al., 2016. Axonal plasticity underpins the functional recovery following surgical decompression in a rat model of cervical spondylotic myelopathy. Acta Neuropathologica Communications 4 89. (10.1186/s40478-016-0359-7)
- Syed, Y. A. et al. 2016. Antibody-mediated neutralization of myelin-associated EphrinB3 accelerates CNS remyelination. Acta Neuropathologica 131 (2), pp.281-298. (10.1007/s00401-015-1521-1)
- Syed, Y. A. et al. 2013. Inhibition of phosphodiesterase-4 promotes oligodendrocyte precursor cell differentiation and enhances CNS remyelination. EMBO Molecular Medicine 5 (12), pp.1918-1934. (10.1002/emmm.201303123)
- Hannan, N. et al., 2013. Generation of multipotent foregut stem cells from human pluripotent stem cells. Stem Cell Reports 1 (4), pp.293-306. (10.1016/j.stemcr.2013.09.003)
- Syed, Y. A. et al. 2011. Inhibition of CNS remyelination by the presence of semaphorin 3A. Journal of Neuroscience 31 (10), pp.3719-3728. (10.1523/JNEUROSCI.4930-10.2011)
- Baer, A. S. et al., 2009. Myelin-mediated inhibition of oligodendrocyte precursor differentiation can be overcome by pharmacological modulation of Fyn-RhoA and protein kinase C signalling. Brain 132 (2), pp.465-481. (10.1093/brain/awn334)
- Syed, Y. A. et al. 2008. Inhibition of oligodendrocyte precursor cell differentiation by myelin-associated proteins. Neurosurgical Focus 24 (3-4) E5. (10.3171/FOC/2008/24/3-4/E4)
Ymchwil
Trosolwg o Ymchwil
Mae anhwylderau niwroddatblygiadol a seiciatrig gan gynnwys anhwylder sbectrwm awtistiaeth (ASD), anabledd deallusol, a sgitsoffrenia yn gyflyrau cymhleth, ac yn aml yn wanhau gyda chanlyniadau cymdeithasol, emosiynol ac economaidd dwfn. Mae datblygiadau diweddar mewn geneteg wedi nodi mwtaniadau risg uchel a newidiadau cromosomaidd mewn loci gan gynnwys 1q21.1, 3q29, a 16p11.2, sy'n cynyddu'n sylweddol sensitifrwydd i'r anhwylderau hyn trwy gydol y cyfnod oes.
Er gwaethaf y darganfyddiadau hyn, mae'r mecanweithiau y mae'r ffactorau risg genetig hyn yn tarfu ar ddatblygiad a swyddogaeth yr ymennydd yn parhau i gael eu deall yn wael. Nod fy ymchwil yw datgelu sail niwrobiolegol risg genetig yn yr anhwylderau hyn, gyda'r nod terfynol o nodi targedau cyffuriau newydd ar gyfer ymyriadau therapiwtig yn y dyfodol.
Amcanion Ymchwil Cyfredol
1. Sail Niwral a Moleciwlaidd Microcephaly a Macrocephaly: Rydym yn astudio annormaleddau maint yr ymennydd sy'n gysylltiedig ag amrywiadau rhif copi 1q21.1 (CNVs), gan ganolbwyntio ar enynnau fel HYDIN2 a NOTCH2NL. Gan ddefnyddio organoidau cortical sy'n deillio o iPSC o unigolion â dileu neu ddyblygu 1q21.1, rydym yn ymchwilio i ddiffygion mewn pensaernïaeth cortical, amlhau niwral, a gwahaniaethu.
2. Mecanweithiau Newidiadau Mater Gwyn mewn Anhwylderau Niwroddatblygiadol: Gan ddefnyddio modelau bôn-gelloedd 2D a 3D o myelination datblygiadol, rydym yn archwilio sut mae CNVs 16p11.2 - sy'n gysylltiedig ag awtistiaeth a chyflyrau seiciatrig - yn tarfu ar uniondeb mater gwyn. Ein nod yw diffinio'r mecanweithiau cellog a moleciwlaidd sy'n sail i myelination wedi'i newid yn ystod datblygiad cynnar yr ymennydd.
3. Cyfraniad Niwrollid i Ddatblygiad yr Ymennydd: Rydym yn archwilio sut mae amlygiad llidiol amenedigol yn rhyngweithio â ffactorau risg genetig i newid trajectories niwro-ddatblygiadol. Gan ddefnyddio astrocytes a microglia sy'n deillio o iPSC o unigolion â CNVs risg uchel, rydym yn asesu newidiadau mewn signalau llidiol a'u heffaith ar swyddogaeth niwronau.
Addysgu
Rwy'n addysgu ar draws rhaglenni israddedig ac ôl-raddedig, ac yn goruchwylio prosiectau traethawd hir blwyddyn olaf ar y ddwy lefel. Mae fy ngoruchwyliaeth yn canolbwyntio'n fras ar bynciau o fewn bioleg ddatblygiadol, niwrowyddoniaeth glinigol, a bioleg gellog.
Rwy'n cyfrannu at y modiwlau canlynol:
- BIT002 Technegau Ymchwil yn y Biowyddorau
- BI3451 Niwrobioleg Anhwylderau'r Ymennydd (Arweinydd Asesu)
- BI3351 Pynciau Cyfoes mewn Clefydau
- BI3001 Prosiect Blwyddyn Olaf Biowyddoniaeth
- BI4001 Prosiect Ymchwil Uwch
- BI4002 Dulliau Ymchwil Uwch
- BI9999 Blwyddyn Hyfforddiant Proffesiynol
Bywgraffiad
Swyddi academaidd
- Uwch Ddarlithydd mewn Niwrowyddoniaeth: Ysgol y Biowyddorau, Prifysgol Caerdydd, Caerdydd
- Arweinydd Grŵp: Sefydliad Niwrowyddoniaeth ac Iechyd Meddwl, Prifysgol Caerdydd, Caerdydd
- Cydymaith Ymchwil Ôl-ddoethurol: Welcome Trust-MRC Sefydliad Bôn-gelloedd Caergrawnt, Prifysgol Caergrawnt, DU
- PhD: Sefydliad Max-Planck ar gyfer Meddygaeth Arbrofol, yr Almaen a Phrifysgol Feddygol Fienna, Awstria.
Aelodaethau proffesiynol
- Yr Academi Addysg Uwch (FHEA)
- Pwyllgor Ymchwil, Sefydliad Peirianneg a Thrwsio Meinwe Caerdydd (CITER)
- Cymdeithas Geneteg, UK
- International Society for Stem Cell Research.
Pwyllgorau ac adolygu
Aelod o'r bwrdd golygyddol
- Ffiniau mewn Niwrowyddoniaeth
- BMC Neurosceince
- Biomed Reseaarch Rhyngwladol
- Journal for Reattach Therapy and Developmental Diversities.
Adolygydd grant
- Swyddi
- ERC
- Cronfa y Cyngor Prydeinig-Newton
- Ymddiriedolaeth Leverhulme
Meysydd goruchwyliaeth
Mae gen i ddiddordeb mewn goruchwylio myfyrwyr PhD yn y meysydd canlynol:
- Bioleg Niwronau a Celloedd Glial
- Anhwylderau Seiciatrig Niwro-Ddatblygiadol
- Niwrollid
- Darganfod Cyffuriau
- Echel Ddatblygiadol y Galon-Ymennydd
Os oes gennych ddiddordeb mewn trafod syniadau, ymuno â'm labordy fel myfyriwr ôl-raddedig neu ôl-ddoethurol, neu wneud cais am gymrodoriaeth, mae croeso i chi gysylltu â mi drwy e-bost
Goruchwyliaeth gyfredol
Ymgysylltu
ArrayContact Details
[email protected]
+44 29206 88314
Adeilad Hadyn Ellis, Llawr 3, Ystafell 3.34a, Heol Maendy, Caerdydd, CF24 4HQ
+44 29206 88314
Adeilad Hadyn Ellis, Llawr 3, Ystafell 3.34a, Heol Maendy, Caerdydd, CF24 4HQ
Themâu ymchwil
Arbenigeddau
- Meddygaeth foleciwlaidd
- Aml-forbidrwydd
- Myelination
- Geneteg Seiciatrig
- Niwroddatblygiad