Professor George Kirov

2025

• Dean, O. et al., 2025. Intravenous ketamine to facilitate transport of agitated patients to the ECT clinic. The Journal of ECT 41 (2), pp.e15-e17. (10.1097/YCT.0000000000001090)
• Dennison, C. A. et al. 2025. Early manifestations of neurodevelopmental copy number variants in children: A population-based investigation. Biological Psychiatry 98 (12), pp.924-933. (10.1016/j.biopsych.2025.03.004)
• Edwards, A. et al. 2025. Large language model–supported identification of intellectual disabilities in clinical free-text summaries: mixed methods study. JMIR AI 4 e72256. (10.2196/72256)
• Gudmundsson, S. et al., 2025. Exploring penetrance of clinically relevant variants in over 800,000 humans from the Genome Aggregation Database. Nature Communications 16 9623. (10.1038/s41467-025-61698-x)
• Katzourou, I. et al. 2025. Contributions of common and rare genetic variation to different measures of mood and anxiety disorder in UK Biobank. BJPsych Open 11 (3) e97. (10.1192/bjo.2025.43)
• Kirov, G. et al. 2025. Risk of dementia after electroconvulsive therapy: a cohort study on the population of Wales. Acta Psychiatrica Scandinavica 152 (4), pp.270-277. (10.1111/acps.70005)
• Koromina, M. et al., 2025. Fine-mapping genomic loci refines bipolar disorder risk genes. Nature Neuroscience 28 (7), pp.1393-1403. (10.1038/s41593-025-01998-z)
• Panagiotaropoulou, G. et al., 2025. Identifying genetic differences between bipolar disorder and major depression through multiple genome-wide association analyses. The British Journal of Psychiatry 226 , pp.79-90. (10.1192/bjp.2024.125)
• Silva, A. et al. 2025. Penetrance of neurodevelopmental copy number variants is associated with variations in cortical morphology. Biological Psychiatry: Cognitive Neuroscience and Neuroimaging 10 (10), pp.1093-1106. (10.1016/j.bpsc.2025.05.010)
• Simmonds, E. et al. 2025. Dementia risk due to traumatic brain injury in subtypes of dementia in the Welsh population. Neurology 105 (3) e213866. (10.1212/wnl.0000000000213866)
• Walker, A. et al., 2025. Genome-wide copy number variation association study in anorexia nervosa. Molecular Psychiatry 30 , pp.2009-2016. (10.1038/s41380-024-02811-2)

2024

• Dinneen, T. J. et al., 2024. Polygenic scores stratify neurodevelopmental copy number variant carrier cognitive outcomes in the UK Biobank. npj Genomic Medicine 9 (1) 43. (10.1038/s41525-024-00426-8)
• Ekstrand, J. et al., 2024. Ketamine or ECT? What have we learned from the KetECT and ELEKT-D trials?. International Journal of Neuropsychopharmacology 27 (1) pyad065. (10.1093/ijnp/pyad065)
• Koromina, M. et al., 2024. Fine-mapping genomic loci refines bipolar disorder risk genes. [Online].medRxiv: OpenRxiv. (10.1101/2024.02.12.24302716)Available at: https://doi.org/10.1101/2024.02.12.24302716.
• Legge, S. E. et al. 2024. Genetic and phenotypic features of Schizophrenia in the UK Biobank. JAMA Psychiatry 81 , pp.681-690. (10.1001/jamapsychiatry.2024.0200)
• Shitomi-Jones, L. M. et al. 2024. Exploration of first onsets of mania, schizophrenia spectrum disorders and major depressive disorder in perimenopause. nature mental health 2 (10), pp.1161-1168. (10.1038/s44220-024-00292-4)
• Stevenson-Hoare, J. et al. 2024. Severe psychiatric disorders are associated with increased risk of dementia. BMJ Mental Health 27 (1) e301097. (10.1136/bmjment-2024-301097)
• Trastulla, L. et al., 2024. Distinct genetic liability profiles define clinically relevant patient strata across common diseases. Nature Communications 15 5534. (10.1038/s41467-024-49338-2)

2023

• Caseras, X. et al. 2023. Copy Number Variants increasing risk for schizophrenia: shared and distinct effects on brain morphometry and cognitive performance. Biological Psychiatry: Global Open Science 3 (4), pp.902-911. (10.1016/j.bpsgos.2022.10.006)
• Lynham, A. J. et al. 2023. DRAGON-Data: A platform and protocol for integrating genomic and phenotypic data across large psychiatric cohorts. BJPsych Open 9 (2) e32. (10.1192/bjo.2022.636)
• Rammos, A. et al. 2023. Family-based analysis of the contribution of rare and common genetic variants to school performance in schizophrenia. Molecular Psychiatry 28 , pp.2081-2087. (10.1038/s41380-023-02013-2)
• Wren, G. et al. 2023. Characterising heart rhythm abnormalities associated with Xp22.31 deletion. Journal of Medical Genetics 60 , pp.636-643. (10.1136/jmg-2022-108862)

2022

• Bracher-Smith, M. et al. 2022. Machine learning for prediction of schizophrenia using genetic and demographic factors in the UK Biobank. Schizophrenia Research 246 , pp.156-164. (10.1016/j.schres.2022.06.006)
• Brcic, L. et al., 2022. Comorbid medical issues in X-linked ichthyosis [Letter]. JID Innovations 2 (3) 100109. (10.1016/j.xjidi.2022.100109)
• Gardner, E. J. et al., 2022. Reduced reproductive success is associated with selective constraint on human genes. Nature 603 (7903), pp.858-863. (10.1038/s41586-022-04549-9)
• Howells, D. et al., 2022. Electroconvulsive therapy reverses cerebral hypoperfusion in a patient with psychotic depression and catatonia. The Journal of ECT 38 (2), pp.141-143. (10.1097/YCT.0000000000000836)
• Singh, T. et al., 2022. Rare coding variants in ten genes confer substantial risk for schizophrenia. Nature 604 , pp.509-516. (10.1038/s41586-022-04556-w)
• Trubetskoy, V. et al., 2022. Mapping genomic loci prioritises genes and implicates synaptic biology in schizophrenia. Nature 604 , pp.502-508. (10.1038/s41586-022-04434-5)

2021

• Caseras, X. et al. 2021. Effects of genomic copy number variants penetrant for schizophrenia on cortical thickness and surface area in healthy individuals: analysis of the UK Biobank. British Journal of Psychiatry 218 (2), pp.104-111. (10.1192/bjp.2020.139)
• Clifton, N. E. et al. 2021. Genetic association of FMRP targets with psychiatric disorders. Molecular Psychiatry 26 , pp.2977-2990. (10.1038/s41380-020-00912-2)
• Hubbard, L. et al. 2021. Rare copy number variations are associated with poorer cognition in schizophrenia. Biological Psychiatry 90 (1), pp.28-34. (10.1016/j.biopsych.2020.11.025)
• Kalman, J. L. et al., 2021. Characterisation of age and polarity at onset in bipolar disorder. The British Journal of Psychiatry 219 (6), pp.659-669. (10.1192/bjp.2021.102)
• Kirov, G. et al. 2021. Electroconvulsive therapy for depression: 80 years of progress. British Journal of Psychiatry 219 (5), pp.594-597. (10.1192/bjp.2021.37)
• Mullins, N. et al., 2021. Genome-wide association study of more than 40,000 bipolar disorder cases provides new insights into the underlying biology. Nature Genetics 53 , pp.817-829. (10.1038/s41588-021-00857-4)
• Rees, E. et al. 2021. Schizophrenia, autism spectrum disorders and developmental disorders share specific disruptive coding mutations. Nature Communications 12 5353. (10.1038/s41467-021-25532-4)
• Rees, E. and Kirov, G. 2021. Copy number variation and neuropsychiatric illness. Current Opinion in Genetics and Development 68 , pp.57-63. (10.1016/j.gde.2021.02.014)
• Silva, A. I. et al. 2021. Analysis of diffusion tensor imaging data from the UK Biobank confirms dosage effect of 15q11.2 copy number variation on white matter and shows association with cognition. Biological Psychiatry 90 (5), pp.307-316. (10.1016/j.biopsych.2021.02.969)

2020

• Akingbuwa, W. A. et al., 2020. Genetic associations between childhood psychopathology and adult depression and associated traits in 42 998 individuals. JAMA Psychiatry 77 (7), pp.715-728. (10.1001/jamapsychiatry.2020.0527)
• Brcic, L. et al. 2020. Medical and neurobehavioural phenotypes in carriers of X-linked ichthyosis-associated genetic deletions in the UK Biobank. Journal of Medical Genetics 57 (10), pp.692-698. (10.1136/jmedgenet-2019-106676)
• Gubb, S. et al. 2020. Medical and neurobehavioural phenotypes in male and female carriers of Xp22.31 duplications in the UK Biobank. Human Molecular Genetics 29 (17), pp.2872-2881. (10.1093/hmg/ddaa174)
• Kendall, K. M. et al. 2020. Impact of schizophrenia genetic liability on the association between schizophrenia and physical illness: a data linkage study. BJPsych Open 6 (6) e139. (10.1192/bjo.2020.42)
• Legge, S. et al. 2020. Clinical indicators of treatment-resistant psychosis. British Journal of Psychiatry 216 (5), pp.259-266. (10.1192/bjp.2019.120)
• Martin, J. et al. 2020. A brief report: de novo copy number variants in children with attention deficit hyperactivity disorder. Translational Psychiatry 10 135. (10.1038/s41398-020-0821-y)
• Rees, E. et al. 2020. De novo mutations identified by exome sequencing implicate rare missense variants in SLC6A1 in schizophrenia. Nature Neuroscience 23 (2), pp.179-184. (10.1038/s41593-019-0565-2)
• Soda, T. et al., 2020. International consortium on the genetics of electroconvulsive therapy and severe depressive disorders (Gen-ECT-ic). European Archives of Psychiatry and Clinical Neuroscience 270 , pp.921-932. (10.1007/s00406-019-01087-w)
• Szatkiewicz, J. P. et al., 2020. Characterization of single gene copy number variants in schizophrenia. Biological Psychiatry 87 (8), pp.736-744. (10.1016/j.biopsych.2019.09.023)
• Warland, A. et al. 2020. Schizophrenia-associated genomic copy number variants and subcortical brain volumes in the UK Biobank. Molecular Psychiatry 25 (4), pp.854-862. (10.1038/s41380-019-0355-y)
• Wong, H. et al., 2020. Contribution of de novo and inherited rare copy number variants to very preterm birth. Journal of Medical Genetics 57 (8), pp.552-557. (10.1136/jmedgenet-2019-106619)

2019

• Crawford, K. et al. 2019. Medical consequences of pathogenic CNVs in adults: Analysis of the UK Biobank. Journal of Medical Genetics 56 , pp.131-138. (10.1136/jmedgenet-2018-105477)
• Escott-Price, V. et al. 2019. Genetic liability to schizophrenia is negatively associated with educational attainment in UK Biobank. Journal of Molecular Psychiatry , pp.-. (10.1038/s41380-018-0328-6)
• Escott-Price, V. et al. 2019. The relationship between common variant schizophrenia liability and number of offspring in the UK Biobank. American Journal of Psychiatry 176 (8), pp.661-666. (10.1176/appi.ajp.2018.18020140)
• Escott-Price, V. et al. 2019. Polygenic risk for schizophrenia and season of birth within the UK Biobank cohort. Psychological Medicine 49 (15), pp.2499-2504. (10.1017/S0033291718000454)
• Kendall, K. M. et al. 2019. Cognitive performance and functional outcomes of carriers of pathogenic copy number variants: analysis of the UK Biobank. British Journal of Psychiatry 214 (05), pp.297-304. (10.1192/bjp.2018.301)
• Kendall, K. M. et al. 2019. Association of rare copy number variants with risk of depression. JAMA Psychiatry 76 (8), pp.818-825. (10.1001/jamapsychiatry.2019.0566)
• Lee, P. H. et al., 2019. Genomic relationships, novel loci, and pleiotropic mechanisms across eight psychiatric disorders. Cell 179 (7), pp.1469-1482.e11. (10.1016/j.cell.2019.11.020)
• Legge, S. E. et al. 2019. Association of genetic liability to psychotic experiences with neuropsychotic disorders and traits. JAMA Psychiatry 76 (12), pp.1256-1265. (10.1001/jamapsychiatry.2019.2508)
• Rees, E. et al. 2019. Targeted sequencing of 10,198 samples confirms abnormalities in neuronal activity and implicates voltage-gated sodium channels in schizophrenia pathogenesis. Biological Psychiatry 85 (7), pp.554-562. (10.1016/j.biopsych.2018.08.022)
• Romagnoni, A. et al., 2019. Comparative performances of machine learning methods for classifying Crohn Disease patients using genome-wide genotyping data. Scientific Reports 9 (1), pp.-. 10351. (10.1038/s41598-019-46649-z)
• Stahl, E. A. et al., 2019. Genome-wide association study identifies 30 loci associated with bipolar disorder. Nature Genetics 51 , pp.793-803. (10.1038/s41588-019-0397-8)
• Vadgama, N. et al., 2019. De novo single-nucleotide and copy number variation in discordant monozygotic twins reveals disease-related genes. European Journal of Human Genetics 27 (7), pp.1121-1133. (10.1038/s41431-019-0376-7)

2018

• Bakhsh, A. D. et al. 2018. An InDel in Phospholipase-C-B-1 is linked with euthyroid multinodular goiter. Thyroid 28 (7), pp.891-901. (10.1089/thy.2017.0312)
• Guyatt, A. L. et al., 2018. Association of copy number variation across the genome with neuropsychiatric traits in the general population. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 177 (5), pp.489-502. (10.1002/ajmg.b.32637)
• Owen, D. et al. 2018. Effects of pathogenic CNVs on physical traits in participants of the UK Biobank. BMC Genomics 19 (1) 867. (10.1186/s12864-018-5292-7)
• Pardinas, A. F. et al. 2018. Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection. Nature Genetics 50 , pp.381-389. (10.1038/s41588-018-0059-2)
• Ruderfer, D. M. et al., 2018. Genomic dissection of bipolar disorder and schizophrenia, including 28 subphenotypes. Cell 173 (7), pp.1705-1715.e16. (10.1016/j.cell.2018.05.046)

2017

• Clifton, N. E. et al. 2017. Schizophrenia copy number variants and associative learning. Molecular Psychiatry 22 (2), pp.178-182. (10.1038/mp.2016.227)
• Green, E. K. et al., 2017. Genome-wide significant locus for Research Diagnostic Criteria Schizoaffective Disorder Bipolar type. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 174 (8), pp.767-771. (10.1002/ajmg.b.32572)
• Kendall, K. , Kirov, G. and Owen, M. 2017. Schizophrenia Genetics. In: Benjamin, S. , Virginia, S. and Pedro, R. eds. Kaplan and Sadock?s Comprehensive Textbook of Psychiatry. Wolters Kluwer
• Kendall, K. M. et al. 2017. Cognitive performance among carriers of pathogenic copy number variants: analysis of 152,000 UK Biobank subjects. Biological Psychiatry 82 (2), pp.P103-110. (10.1016/j.biopsych.2016.08.014)
• Legge, S. E. et al. 2017. Genome-wide common and rare variant analysis provides novel insights into clozapine-associated neutropenia. Molecular Psychiatry 22 , pp.1502-1508. (10.1038/mp.2016.97)
• Marshall, C. R. et al., 2017. Contribution of copy number variants to schizophrenia from a genome-wide study of 41,321 subjects. Nature Genetics 49 , pp.27-35. (10.1038/ng.3725)
• McLaughlin, R. L. et al., 2017. Genetic correlation between amyotrophic lateral sclerosis and schizophrenia. Nature Communications 8 14774. (10.1038/ncomms14774)
• Singh, T. et al., 2017. The contribution of rare variants to risk of schizophrenia in individuals with and without intellectual disability. Nature Genetics 49 , pp.1167-1173. (10.1038/ng.3903)
• Witt, S. H. et al., 2017. Genome-wide association study of borderline personality disorder reveals genetic overlap with the bipolar disorder, schizophrenia and major depression. Translational Psychiatry 7 e1155.

2016

• Bigdeli, T. B. et al., 2016. Genome-wide association study reveals greater polygenic loading for schizophrenia in cases with a family history of illness. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 171 (2), pp.276-289. (10.1002/ajmg.b.32402)
• Carroll, L. S. et al., 2016. Mutation screening of SCN2A in schizophrenia and identification of a novel loss-of-function mutation. Psychiatric Genetics 26 (2), pp.60-65. (10.1097/YPG.0000000000000110)
• Franke, B. et al., 2016. Genetic influences on schizophrenia and subcortical brain volumes: large-scale proof of concept. Nature Neuroscience 19 (3), pp.420-431. (10.1038/nn.4228)
• Fry, A. E. et al. 2016. Pathogenic copy number variants and SCN1A mutations in patients with intellectual disability and childhood-onset epilepsy. BMC Medical Genetics 17 , pp.-. 34. (10.1186/s12881-016-0294-2)
• Han, J. et al. 2016. Gender differences in CNV burden do not confound schizophrenia CNV associations. Scientific Reports 6 25986. (10.1038/srep25986)
• Isles, A. R. et al. 2016. Parental origin of interstitial duplications at 15q11.2-q13.3 in schizophrenia and neurodevelopmental disorders. PLoS Genetics 12 (5) e1005993. (10.1371/journal.pgen.1005993)
• Kirov, G. G. et al. 2016. Evaluation of cumulative cognitive deficits from electroconvulsive therapy. British Journal of Psychiatry 208 (3), pp.266-270. (10.1192/bjp.bp.114.158261)
• Pardinas, A. et al. 2016. Common schizophrenia alleles are enriched in mutation-intolerant genes and maintained by background selection. [Online].bioRxiv. (10.1101/068593)Available at: http://dx.doi.org/10.1101/068593.
• Rees, E. et al. 2016. Analysis of intellectual disability copy number variants for association with schizophrenia. JAMA Psychiatry 73 (9), pp.963-969. (10.1001/jamapsychiatry.2016.1831)
• Singh, T. et al., 2016. Rare loss-of-function variants in KMT2F are associated with schizophrenia and developmental disorders. Nature Neuroscience 19 , pp.571-577. (10.1101/036384)
• Singh, T. et al., 2016. Rare loss-of-function variants in SETD1A are associated with schizophrenia and developmental disorders. Nature Neuroscience 19 (4), pp.571-577. (10.1038/nn.4267)
• Tansey, K. E. et al. 2016. Common alleles contribute to schizophrenia in CNV carriers. Molecular Psychiatry 21 , pp.1085-1089. (10.1038/mp.2015.143)

2015

• Bulik-Sullivan, B. K. et al., 2015. LD Score regression distinguishes confounding from polygenicity in genome-wide association studies. Nature Genetics 47 (3), pp.291-295. (10.1038/ng.3211)
• Escott-Price, V. et al. 2015. No evidence for enrichment in schizophrenia for common allelic associations at imprinted loci. PLoS ONE 10 (12), pp.-. e0144172. (10.1371/journal.pone.0144172)
• Green, E. K. et al., 2015. Copy number variation in bipolar disorder. Molecular Psychiatry 21 (1), pp.89-93. (10.1038/mp.2014.174)
• Kirov, G. 2015. CNVs in neuropsychiatric disorders. Human Molecular Genetics 24 (R1), pp.R45-R49. (10.1093/hmg/ddv253)
• Kirov, G. , Rees, E. and Walters, J. T. R. 2015. What a psychiatrist needs to know about copy number variants. BJPscyh Advances 21 (3), pp.157-163. (10.1192/apt.bp.113.012039)
• Lee, S. H. et al., 2015. New data and an old puzzle: the negative association between schizophrenia and rheumatoid arthritis. International Journal of Epidemiology 44 (5), pp.1706-1721. (10.1093/ije/dyv136)
• Maier, R. et al., 2015. Joint analysis of psychiatric disorders increases accuracy of risk prediction for schizophrenia, bipolar disorder, and major depressive disorder. American Journal of Human Genetics 96 (2), pp.283-294. (10.1016/j.ajhg.2014.12.006)
• O'Dushlaine, C. et al., 2015. Psychiatric genome-wide association study analyses implicate neuronal, immune and histone pathways. Nature Neuroscience 18 (2), pp.199-209. (10.1038/nn.3922)
• Pocklington, A. et al. 2015. Novel findings from CNVs implicate inhibitory and excitatory signaling complexes in schizophrenia. Neuron 86 (5), pp.1203-1214. (10.1016/j.neuron.2015.04.022)
• Rees, E. et al. 2015. Analysis of exome sequence in 604 trios for recessive genotypes in schizophrenia. Translational Psychiatry 5 (7) e607. (10.1038/tp.2015.99)
• Tansey, K. E. et al. 2015. Common alleles contribute to schizophrenia in CNV carriers [Erratum]. Molecular Psychiatry 21 1153. (10.1038/mp.2015.170)

2014

• Fromer, M. et al., 2014. De novo mutations in schizophrenia implicate synaptic networks. Nature 506 , pp.179-184. (10.1038/nature12929)
• Georgieva, L. et al. 2014. De novo CNVs in bipolar affective disorder and schizophrenia. Human Molecular Genetics 23 (24), pp.6677-6683. (10.1093/hmg/ddu379)
• Gusev, A. et al., 2014. Partitioning heritability of regulatory and cell-type-specific variants across 11 common diseases. American Journal of Human Genetics 95 (5), pp.535-552. (10.1016/j.ajhg.2014.10.004)
• Hoeffding, L. K. E. et al., 2014. Sequence analysis of 17NRXN1deletions. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 165 (1), pp.52-61. (10.1002/ajmg.b.32204)
• Morris, D. W. et al., 2014. An inherited duplication at the gene p21 Protein-Activated Kinase 7 (PAK7) is a risk factor for psychosis. Human Molecular Genetics 23 (12), pp.3316-3326. (10.1093/hmg/ddu025)
• Mulle, J. G. et al., 2014. Reciprocal duplication of the Williams-Beuren Syndrome deletion on chromosome 7q11.23 is associated with schizophrenia. Biological Psychiatry 75 (5), pp.371-7. (10.1016/j.biopsych.2013.05.040)
• Peall, K. J. et al. 2014. SGCE and myoclonus dystonia: motor characteristics, diagnostic criteria and clinical predictors of genotype. Journal of Neurology 261 (12), pp.2296-2304. (10.1007/s00415-014-7488-3)
• Rees, E. et al. 2014. CNV analysis in a large schizophrenia sample implicates deletions at 16p12.1 and SLC1A1 and duplications at 1p36.33 and CGNL1. Human Molecular Genetics 23 (6), pp.1669-1676. (10.1093/hmg/ddt540)
• Rees, E. et al. 2014. Analysis of copy number variations at 15 schizophrenia-associated loci. British Journal of Psychiatry 204 (2), pp.108-114. (10.1192/bjp.bp.113.131052)
• Rees, E. et al. 2014. Authors' reply [Letter]. British Journal of Psychiatry 205 (1), pp.78. (10.1192/bjp.205.1.78)
• Ripke, S. et al., 2014. Biological insights from 108 schizophrenia-associated genetic loci. Nature 511 (7510), pp.421-427. (10.1038/nature13595)
• Szatkiewicz, J. P. et al., 2014. Copy number variation in schizophrenia in Sweden. Molecular Psychiatry 19 (7), pp.762-773. (10.1038/mp.2014.40)

2013

• Aberg, K. A. et al., 2013. A comprehensive family-based replication study of schizophrenia genes. JAMA Psychiatry 70 (6), pp.573-581. (10.1001/jamapsychiatry.2013.288)
• Betcheva, E. et al., 2013. Whole-genome-wide association study in the Bulgarian population reveals HHAT as schizophrenia susceptibility gene. Psychiatric Genetics 23 (1), pp.11-19. (10.1097/YPG.0b013e3283586343)
• Betcheva, E. T. et al., 2013. Whole-genome-wide association study in the Bulgarian population reveals HHAT as schizophrenia susceptibility gene [Article]. Psychiatric Genetics 23 (1), pp.11-19. (10.1097/YPG.0b013e3283586343)
• Chapman, J. et al., 2013. A genome-wide study shows a limited contribution of rare copy number variants to Alzheimer's disease risk. Human Molecular Genetics 22 (4), pp.816-824. (10.1093/hmg/dds476)
• Green, E. K. et al. 2013. Replication of bipolar disorder susceptibility alleles and identification of two novel genome-wide significant associations in a new bipolar disorder case-control sample. Molecular Psychiatry 18 (12), pp.1302-1307. (10.1038/mp.2012.142)
• Grozeva, D. et al. 2013. Reduced burden of very large and rare CNVs in bipolar affective disorder. Bipolar Disorders 15 (8), pp.893-8. (10.1111/bdi.12125)
• Guha, S. et al., 2013. Implication of a rare deletion at distal 16p11.2 in schizophrenia. JAMA Psychiatry 70 (3), pp.253-260. (10.1001/2013.jamapsychiatry.71)
• Kim, Y. et al., 2013. Non-random mating, parent-of-origin, and maternal-fetal incompatibility effects in schizophrenia. Schizophrenia Research 143 (1), pp.11-17. (10.1016/j.schres.2012.11.002)
• Kirov, G. et al. 2013. The penetrance of copy number variations for schizophrenia and developmental delay. Biological Psychiatry 75 (5), pp.378-385. (10.1016/j.biopsych.2013.07.022)
• Kronenberg, F. et al., 2013. Novel Loci Associated with Increased Risk of Sudden Cardiac Death in the Context of Coronary Artery Disease [Article]. PLoS ONE 8 (4), pp.e59905. (10.1371/journal.pone.0059905)
• Lee, S. et al., 2013. Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs. Nature Genetics 45 (9), pp.984-994. (10.1038/ng.2711)
• Lionel, A. C. et al., 2013. Rare exonic deletions implicate the synaptic organizer Gephyrin (GPHN) in risk for autism, schizophrenia and seizures. Human Molecular Genetics 22 (10), pp.2055-2066. (10.1093/hmg/ddt056)
• Peall, K. et al., 2013. Are psychiatric symptoms a core phenotype of myoclonus dystonia syndrome caused by SGCE mutations?. Journal of Neurology, Neurosurgery & Psychiatry 84 (9) e1. (10.1136/jnnp-2013-306103.24)
• Peall, K. J. et al. 2013. SGCE mutations cause psychiatric disorders: clinical and genetic characterization. Brain 136 (1), pp.294-303. (10.1093/brain/aws308)
• Rees, E. et al. 2013. Evidence that duplications of 22q11.2 protect against schizophrenia. Molecular Psychiatry n/a (10.1038/mp.2013.156)
• Ruderfer, D. et al., 2013. Mosaic copy number variation in schizophrenia. European Journal of Human Genetics 21 (9), pp.1007-1011. (10.1038/ejhg.2012.287)
• Terwisscha van Scheltinga, A. F. et al., 2013. Genetic schizophrenia risk variants jointly modulate total brain and white matter volume. Biological Psychiatry 73 (6), pp.525-531. (10.1016/j.biopsych.2012.08.017)
• van Scheltinga, A. F. T. et al., 2013. Schizophrenia genetic variants are not associated with intelligence. Psychological Medicine 43 (12), pp.2563-2570. (10.1017/S0033291713000196)
• Williams, H. J. et al. 2013. Schizophrenia two-hit hypothesis in velo-cardio facial syndrome. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 162 (2), pp.177-182. (10.1002/ajmg.b.32129)

2012

• Badner, J. A. et al., 2012. Genome-wide linkage analysis of 972 bipolar pedigrees using single-nucleotide polymorphisms. Molecular Psychiatry 17 (8), pp.818-826. (10.1038/mp.2011.89)
• Cooper, J. D. et al., 2012. Seven newly identified loci for autoimmune thyroid disease. Human Molecular Genetics 21 (23), pp.5202-5208. (10.1093/hmg/dds357)
• Derks, E. M. et al., 2012. Investigation of the genetic association between quantitative measures of psychosis and schizophrenia: A polygenic risk score analysis. PLoS ONE 7 (6) e37852. (10.1371/journal.pone.0037852)
• Forstbauer, L. M. et al., 2012. Genome-wide pooling approach identifies SPATA5 as a new susceptibility locus for alopecia areata. European Journal of Human Genetics 20 (3), pp.326-332. (10.1038/ejhg.2011.185)
• Fromer, M. et al., 2012. Discovery and statistical genotyping of copy-number variation from whole-exome sequencing depth. American Journal of Human Genetics 91 (4), pp.597-607. (10.1016/j.ajhg.2012.08.005)
• Grozeva, D. et al. 2012. Independent estimation of the frequency of rare CNVs in the UK population confirms their role in schizophrenia. Schizophrenia Research 135 (1-3), pp.1-7. (10.1016/j.schres.2011.11.004)
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2011

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2010

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2009

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2008

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2007

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2006

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2005

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2004

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• Williams, N. M. et al. 2004. Support for RGS4 as a susceptibility gene for schizophrenia. Biological Psychiatry 55 (2), pp.192-195. (10.1016/j.biopsych.2003.11.002)

2003

• Børglum, A. et al., 2003. Possible parent-of-origin effect of Dopa decarboxylase in susceptibility to bipolar affective disorder. American Journal of Medical Genetics 117B (1), pp.18-22. (10.1002/ajmg.b.10030)
• Farbrother, J. E. et al., 2003. Linkage analysis of 18p, 12q and 17q high myopia loci in 51 UK families. Investigative ophthalmology & visual science 44 (E-Abst)
• Georgieva, L. et al. 2003. Genetic variation in the seven-pass transmembrane cadherin CELSR1. Psychiatric Genetics 13 (2), pp.103-106. (10.1097/01.ypg.0000057486.14812.03)
• Ivanov, D. et al. 2003. Chromosome 22q11 deletions, velo-cardio-facial syndrome and early-onset psychosis: Molecular genetic study. The British Journal of Psychiatry 183 (5), pp.409-413. (10.1192/bjp.183.5.409)
• Kirov, G. et al. 2003. Association analysis of the HOPA12bp polymorphism in schizophrenia and manic depressive illness. American Journal of Medical Genetics 118B (1), pp.16-19. (10.1002/ajmg.b.10065)
• Kirov, G. et al. 2003. Variation in the protocadherin - A gene cluster?. Genomics 82 (4), pp.433-440. (10.1016/S0888-7543(03)00167-8)
• Kirov, G. and Korszun, A. 2003. The antidepressant debate should move on. British Journal of Psychiatry 182 (6), pp.551-551. (10.1192/bjp.182.6.551)
• Williams, H. J. et al., 2003. Association between PRODH and schizophrenia is not confirmed. Molecular Psychiatry 8 (7), pp.644-645. (10.1038/sj.mp.4001276)
• Williams, H. et al., 2003. Detailed analysis of PRODH and PsPRODH reveals no association with schizophrenia. American Journal of Medical Genetics 120B (1), pp.42-46. (10.1002/ajmg.b.20049)

2002

• Bennett, P. et al., 2002. The Wellcome trust UK-Irish bipolar affective disorder sibling-pair genome screen: first stage report. Molecular Psychiatry 7 (2), pp.189-200. (10.1038/sj.mp.4000957)
• Bray, N. J. et al. 2002. Screening the human protocadherin 8 (PCDH8) gene in schizophrenia. Genes, Brain and Behavior 1 (3), pp.187-191. (10.1034/j.1601-183X.2002.10307.x)
• Bray, N. J. et al. 2002. Screening the protocadherin 8 (PCDH8) gene in schizophrenia [Conference Abstract]. American Journal of Medical Genetics 114 (7), pp.844-844.
• Dimitrova, A. et al., 2002. Major psychiatric disorders and the serotonin transporter gene (SLC6A4): family-based association studies. Psychiatric Genetics 12 (3), pp.137-141. (10.1097/00041444-200209000-00004)
• Georgieva, L. et al. 2002. Dopamine transporter gene (DAT1) VNTR polymorphism in major psychiatric disorders: family-based association study in the Bulgarian population. Acta Psychiatrica Scandinavica 105 (5), pp.396-399. (10.1034/j.1600-0447.2002.1o174.x)
• Kirov, G. 2002. Use of a lithium analyzer machine in a mood disorders clinics. Journal of affective disorders 68 (1), pp.120-120.
• Norton, N. et al., 2002. Schizophrenia and functional polymorphisms in the MAOA and COMT genes: No evidence for association or epistasis. American Journal Of Medical Genetics Part A 114 (5), pp.491-496. (10.1002/ajmg.10517)
• Norton, N. et al., 2002. Universal, robust, highly quantitative SNP allele frequency measurement in DNA pools. Human Genetics 110 (5), pp.471-478. (10.1007/s00439-002-0706-6)
• O'Mahony, E. et al., 2002. Sibling pairs with affective disorders: resemblance of demographic and clinical features. Psychological Medicine 32 (1), pp.55-61. (10.1017/S0033291701004986)
• Sklar, P. et al., 2002. Family-based association study of 76 candidate genes in bipolar disorder: BDNF is a potential risk locus. Molecular Psychiatry 7 (6), pp.579. (10.1038/sj.mp.4001058)
• Williams, N. M. et al. 2002. Mutation screening and LD mapping in the VCFS deleted region of chromosome 22q11 in schizophrenia using a novel DNA pooling approach. Molecular Psychiatry 7 (10), pp.1092-1100. (10.1038/sj.mp.4001188)

2001

• Kirov, G. et al. 2001. Screening ABCG1, the human homologue of the Drosophila white gene, for polymorphisms and association with bipolar affective disorder. Molecular Psychiatry 6 (6), pp.671-677. (10.1038/sj.mp.4000899)
• Kirov, G. , Stehens, M. and Owen, M. J. 2001. Screening of three candidate genes in the bipolar candidate region on chromosome 21q22.3. American Journal of Medical Genetics 105 (7), pp.612-612.
• Stephens, M. , Kirov, G. and Owen, M. J. 2001. ADARB1, a brain specific member of the RNA-specific adenosine deaminase family and bipolar affective disorder. American Journal of Medical Genetics 105 (7), pp.622-622.
• Williams, H. et al., 2001. Further analysis of KIAA0027 in schizophrenic patients. American Journal of Medical Genetics 105 (7), pp.561-561.

2000

• Bowen, T. et al. 2000. No evidence of association from transmission disequilibrium analysis of the hKCa3 gene in bipolar disorder. Bipolar Disorders 2 (4), pp.328-331. (10.1034/j.1399-5618.2000.020406.x)
• Guggenheim, J. A. , Kirov, G. and Hodson, S. A. 2000. The heritability of high myopia: a reanalysis of Goldschmidt's data. Journal of Medical Genetics 37 (3), pp.227-231. (10.1136/jmg.37.3.227)
• Hoogendoorn, B. et al. 2000. Cheap, accurate and rapid allele frequency estimation of single nucleotide polymorphisms by primer extension and DHPLC in DNA pools. Human Genetics 107 (5), pp.488-493. (10.1007/s004390000397)
• Kirov, G. et al. 2000. Comparison between clinical and demographic characteristics of bipolar patients collected in two different countries.. American Journal of Medical Genetics 96 (4), pp.506-506.
• Kirov, G. , Stephens, M. and Owen, M. J. 2000. Examining the 4p16 region for association with bipolar disorder using DNA pooling. American Journal of Medical Genetics 96 (4), pp.548-548.
• Kirov, G. et al. 2000. Automated genotyping of single-nucleotide polymorphisms by extension of fluorescently labelled primers: analysis of individual and pooled DNA samples. Balkan Journal of Medical Genetics 3 , pp.23-28.
• Kirov, G. et al. 2000. Pooled genotyping of microsatellite markers in parent-offspring trios. Genome Research 10 (1), pp.105-115.
• O'Mahony, E. et al., 2000. Clinical similarities in siblings with bipolar disorder. American Journal of Medical Genetics 96 (4), pp.514-514.

1999

• Birkett, J. T. P. et al., 1999. Association between a 2030-G/A substitution in the human myo-inositol monophosphatase gene (IMPA) and bipolar disorder and schizophrenia. Molecular Psychiatry 4 , pp.S103-S103.
• Bolonna, A. A. et al., 1999. An investigation of a structural polymorphism in the mGluR7 gene in association with schizophrenia. Schizophrenia Research 36 (1-3), pp.99-99.
• Borglum, A. D. et al., 1999. Association study of the DOPA decarboxylase gene in bipolar affective disorder and schizophrenia. Molecular Psychiatry 4 , pp.S103-S104.
• Borglum, A. D. et al., 1999. Two novel variants in the DOPA decarboxylase gene: association with bipolar affective disorder. Molecular Psychiatry 4 (6), pp.545-551. (10.1038/sj.mp.4000559)
• Bowen, T. et al. 1999. Linkage studies of bipolar disorder with chromosome 18 markers. American Journal of Medical Genetics 15 (88), pp.503-509. (10.1002/(SICI)1096-8628(19991015)88:5<503::AID-AJMG13>3.0.CO;2-U)
• Kirov, G. et al. 1999. Family-based association studies of bipolar disorder with candidate genes involved in dopamine neurotransmission: DBH, DAT1, COMT, DRD2, DRD3 and DRD5. Molecular Psychiatry 4 (6), pp.558-565.
• Kirov, G. and Murray, R. M. 1999. Ethnic differences in the presentation of bipolar affective disorder. European Psychiatry 14 (4), pp.199-204.
• Kirov, G. et al. 1999. A functional polymorphism in the promoter of monoamine oxidase A gene and bipolar affective disorder. International Journal of Neuropsychopharmacology 2 (4), pp.293-298. (10.1017/s1461145799001601)
• Kirov, G. and Owen, M. J. 1999. Identification of new microsatellite markers on chromosome 4p16. American Journal of Human Genetics 65 (4), pp.A257-A257.
• Kirov, G. et al. 1999. Tryptophan hydroxylase gene and manic-depressive illness [letter]. Archives of General Psychiatry 56 (1), pp.98-99. (10.1001/archpsyc.56.1.98)
• Kirov, G. et al. 1999. Bipolar disorder and the serotonin transporter gene: a family-based association study. Psychological Medicine -London- 29 (5), pp.1249-1254. (10.1017/s003329179900882x)
• Li, T. et al., 1999. Association analysis between dopamine receptor genes and bipolar affective disorder. Psychiatry Research 86 (3), pp.193-201. (10.1016/S0165-1781(99)00034-7)
• Petouni, M. et al., 1999. Association study between genetic variants in the alpha- adrenergic receptor genes and bipolar affective disorder. Molecular Psychiatry 4 , pp.S112-S112.
• Williams, N. M. et al. 1999. Polymorphism screening and association studies of the Wolfram syndrome gene in schizophrenia. Molecular Psychiatry 4 (supp1), pp.S42-S42.

1998

• Arranz, M. J. et al., 1998. Genetic variation in the 5-HT3 receptor gene: No association with schizophrenia or drug response. Schizophrenia Research 29 (1-2), pp.127-128.
• Arranz, M. J. et al., 1998. A polymorphism in the promoter region of the dopamine D2 receptor gene (DRD2) and drug response: Association studies. Schizophrenia Research 29 (1-2), pp.127-127.
• Arranz, M. J. et al., 1998. Evidence for association between polymorphisms in the promoter and coding regions of the 5-HT2A receptor gene and response to clozapine. Molecular Psychiatry 3 (1), pp.61-66. (10.1038/sj.mp.4000348)
• Arranz, M. J. et al., 1998. Meta-analysis of studies on genetic variation in 5-HT2A receptors and clozapine response. Schizophrenia Research 32 (2), pp.93-99. (10.1016/S0920-9964(98)00032-2)
• Birkett, J. T. P. et al., 1998. No association between a variable number of tandem repeat (VNTR) in the 3 ' untranslated region of the dopamine transporter gene (DAT1) and bipolar affective disorder. American Journal of Medical Genetics 81 (6), pp.500-501.
• David, A. S. et al., 1998. Improving insight and compliance: Predictions and consequences. Schizophrenia Research 29 (1-2), pp.34-35. (10.1016/S0920-9964(97)88375-2)
• Healey, A. et al., 1998. Cost-effectiveness evaluation of compliance therapy for people with psychosis. British Journal of Psychiatry 172 , pp.420-424. (10.1192/bjp.172.5.420)
• Holmes, C. et al., 1998. Apolipoprotein E: depressive illness, depressive symptoms, and Alzheimer's disease. Biological psychiatry 43 (3), pp.159-164. (10.1016/S0006-3223(97)00326-0)
• Kaneva, R. et al., 1998. A linkage study of affective disorders in two Bulgarian Gypsy families: results for candidate regions on chromosomes 18 and 21. Psychiatric Genetics 8 (4), pp.245-249. (10.1097/00041444-199808040-00008)
• Kemp, R. et al., 1998. Randomised controlled trial of compliance therapy. 18-month follow-up. British Journal of Psychiatry 172 , pp.413-419. (10.1192/bjp.172.5.413)
• Kirov, G. 1998. Thyroid disorders in lithium-treated patients. Journal of affective disorders 50 (1), pp.33-40.
• Kirov, G. et al. 1998. Family-based association studies of candidate genes in bipolar disorder. American Journal of Medical Genetics 81 (6), pp.476-477.
• Kirov, G. et al. 1998. Religious faith after psychotic illness. Psychopathology 31 (5), pp.234-245. (10.1159/000029045)
• Kirov, G. et al. 1998. Low activity allele of catechol-O-methyltransferase gene associated with rapid cycling bipolar disorder. Molecular Psychiatry 3 (4), pp.342-345. (10.1038/sj.mp.4000385)
• Padareva, V. et al., 1998. Modification of blowing agent system based on sodium bicarbonate with activated natural zeolite. Journal of Materials Science Letters 17 (2), pp.107-109.
• Vallada, H. P. et al., 1998. Linkage analysis between bipolar affective disorder and markers on chromosome X. Psychiatric Genetics 8 (3), pp.183-186. (10.1097/00041444-199800830-00008)
• Williams, N. M. et al. 1998. Screening the critical region on chromosome 4p with DNA pooling for association with bipolar disorder. American Journal of Medical Genetics 81 (6), pp.486-486.

1997

• Arranz, M. J. et al., 1997. 5-HT2A receptor and bipolar affective disorder: association studies in affected patients. Neuroscience Letters 224 (2), pp.95-98. (10.1016/S0304-3940(97)13456-5)
• Arranz, M. J. et al., 1997. Novel polymorphisms detected in the 5-HT3 receptor gene: Association studies with schizophrenia and clozapine response. American Journal of Medical Genetics 74 (6), pp.615-615.
• Arranz, M. et al., 1997. Polymorphisms in the 5-HT2A receptor gene and promoter region associated with clozapine response. Schizophrenia Research 24 (1-2), pp.90. (10.1016/S0920-9964(97)82243-8)
• Bolonna, A. A. et al., 1997. Analysis of the NMDA receptor in schizophrenia. American Journal of Medical Genetics 74 (6), pp.631-631.
• Bolonna, A. et al., 1997. An investigation of the GLUR6 receptor gene (GRIK2) in schizophrenia. Schizophrenia Research 24 (1-2), pp.93. (10.1016/S0920-9964(97)82252-9)
• Craddock, N. J. et al. 1997. Dimensional classification for use in biological studies of bipolar disorder. American Journal of Medical Genetics 74 (6), pp.588-588.
• Dawson, E. et al., 1997. The brain cannabinoid receptor gene and bipolar affective disorders. American Journal of Medical Genetics 74 (6), pp.613-614.
• Kirov, G. and Murray, R. 1997. The molecular genetics of schizophrenia: progress so far. Molecular Medicine Today 3 (3), pp.124-130. (10.1016/S1357-4310(96)10060-5)
• Kirov, G. et al. 1997. Observations on switching patients with schizophrenia to risperidone treatment. Risperidone Switching Study Group. Acta Psychiatrica Scandinavica 95 (5), pp.439-443. (10.1111/j.1600-0447.1997.tb09659.x)
• Kunugi, H. et al., 1997. No evidence for an association of affective disorders with high- or low-activity allele of catechol-o-methyltransferase gene. Biological psychiatry 42 (4), pp.282-285. (10.1016/S0006-3223(96)00366-6)
• Li, T. et al., 1997. Allelic functional variation of serotonin transporter expression is a susceptibility factor for late onset Alzheimer's disease. NeuroReport 8 (3), pp.683-686. (10.1097/00001756-199702100-00021)
• Sodhi, M. S. et al., 1997. Allelic variation of the 5-HT2C receptor in psychosis. Schizophrenia Research 24 (1-2), pp.90. (10.1016/S0920-9964(97)82246-3)
• Vallada, H. P. et al., 1997. Linkage studies in bipolar affective disorder with markers on chromosome Xq25-27. American Journal of Medical Genetics 74 (6), pp.679-680.

1996

• Arranz, M. J. et al., 1996. Analysis of a structural polymorphism in the 5-HT2A receptor and clinical response to clozapine. Neuroscience Letters 217 (2-3), pp.177-178. (10.1016/0304-3940(96)13094-9)
• Arranz, M. et al., 1996. 5-HT2A polymorphisms and mental disorders. Schizophrenia Research 18 (2-3), pp.164. (10.1016/0920-9964(96)85523-X)
• Collier, D. A. et al., 1996. The serotonin transporter is a potential susceptibility factor for bipolar affective disorder. NeuroReport 7 (10), pp.1675-1679. (10.1097/00001756-199607080-00030)
• Collier, D. A. et al., 1996. A novel functional polymorphism within the promoter of the serotonin transporter gene: possible role in susceptibility to affective disorders. Molecular Psychiatry 1 (6), pp.453-460.
• Kemp, R. et al., 1996. A randomised controlled trial of compliance therapy in psychosis: Follow-up results and predictors of outcome. Schizophrenia Research 18 (2-3), pp.135. (10.1016/0920-9964(96)85452-1)
• Kirov, G. et al. 1996. Do obstetric complications cause the earlier age at onset in male than female schizophrenics?. Schizophrenia Research 20 (1-2), pp.117-124. (10.1016/0920-9964(95)00063-1)
• Sodhi, M. S. et al., 1996. Association between psychoses and allelic variation in the 5-HT2C receptor gene. Schizophrenia Research 18 (2-3), pp.IXB2.
• Vallada, H. et al., 1996. Linkage studies in bipolar affective disorder with markers on chromosome 21. Journal of affective disorders 41 (3), pp.217-221. (10.1016/s0165-0327(96)00055-9)

1994

• Kirov, G. et al. 1994. Plasma magnesium levels in a population of psychiatric patients: correlations with symptoms. Neuropsychobiology 30 (2-3), pp.73-78. (10.1159/000119139)
• Kirov, G. , Jones, P. and Lewis, S. 1994. Prevalence of Delusional Misidentification Syndromes. Psychopathology 27 (3-5), pp.148-149. (10.1159/000284862)
• Kirov, G. , Jones, P. and Murray, R. 1994. Disappearance of gender differences in age at onset in schizophrenics with a familial loading. Acta Psychiatrica Scandinavica 90 (3), pp.236-237. (10.1111/j.1600-0447.1994.tb01583.x)

1990

• Kirov, G. 1990. Slowly progressive schizophrenia. British Journal of Psychiatry 157 , pp.457-457.
• Kirov, G. and Tsachev, K. N. 1990. Magnesium, Schizophrenia and Manic-Depressive Disease. Neuropsychobiology 23 (2), pp.79-81. (10.1159/000119431)

1987

• Nikolova, S. et al., 1987. [In vitro research on the interaction of natural zeolites with diploid cells from the human embryonic lung].. Problemi Na Khigienata 12 , pp.133-141.