Professor Sir Michael Owen

BSc, MB ChB, PhD, FRCPsych, FMedSci, FLSW

2021

• Cleynen, I. et al., 2021. Genetic contributors to risk of schizophrenia in the presence of a 22q11.2 deletion. Molecular Psychiatry , pp.4496-4510. (10.1038/s41380-020-0654-3)
• Cunningham, A. et al. 2021. Coordination difficulties, IQ and psychopathology in children with high-risk Copy Number Variants. Psychological Medicine 51 (2), pp.290-299. (10.1017/S0033291719003210)

2020

• Glanville, K. P. et al., 2020. Classical human leukocyte antigen alleles and C4 haplotypes are not significantly associated with depression. Biological Psychiatry 87 (5), pp.419-430. (10.1016/j.biopsych.2019.06.031)
• Hall, L. S. et al. 2020. A transcriptome-wide association study implicates specific pre- and post-synaptic abnormalities in schizophrenia. Human Molecular Genetics 29 (1), pp.159-167. (10.1093/hmg/ddz253)
• Legge, S. et al. 2020. Clinical indicators of treatment-resistant psychosis. British Journal of Psychiatry 216 (5), pp.259-266. (10.1192/bjp.2019.120)
• Lewis, K. J. et al. 2020. Comparison of genetic liability for sleep traits among individuals with Bipolar Disorder I or II and control participants. JAMA Psychiatry 77 (3), pp.303-310. (10.1001/jamapsychiatry.2019.4079)
• Moulding, H. et al. 2020. Sleep problems and associations with psychopathology and cognition in young people with 22q11.2 deletion syndrome (22q11.2DS). Psychological Medicine 50 , pp.1191-1202. (10.1017/S0033291719001119)
• Rees, E. et al. 2020. De novo mutations identified by exome sequencing implicate rare missense variants in SLC6A1 in schizophrenia. Nature Neuroscience 23 (2), pp.179-184. (10.1038/s41593-019-0565-2)
• Richards, A. et al. 2020. The relationship between polygenic risk scores and cognition in schizophrenia. Schizophrenia Bulletin 46 (2), pp.-. (10.1093/schbul/sbz061)
• Sullivan, S. A. et al., 2020. A population-based cohort study examining the incidence and impact of psychotic experiences from childhood to adulthood, and prediction of psychotic disorder. American Journal of Psychiatry 177 (4), pp.308-317. (10.1176/appi.ajp.2019.19060654)
• Sun, D. et al., 2020. Large-scale mapping of cortical alterations in 22q11.2 deletion syndrome: Convergence with idiopathic psychosis and effects of deletion size. Molecular Psychiatry 25 , pp.1822-1834. (10.1038/s41380-018-0078-5)
• Szatkiewicz, J. P. et al., 2020. Characterization of single gene copy number variants in schizophrenia. Biological Psychiatry 87 (8), pp.736-744. (10.1016/j.biopsych.2019.09.023)
• Villalon-Reina, J. et al., 2020. Altered white matter microstructure in 22q11.2 deletion syndrome: a multi-site diffusion tensor imaging study. Molecular Psychiatry 25 , pp.2818-2831. (10.1038/s41380-019-0450-0)

2019

• Chapman, R. M. et al., 2019. Convergent evidence that ZNF804A is a regulator of pre-messenger RNA processing and gene expression. Schizophrenia Bulletin 45 (6), pp.1267-1278. (10.1093/schbul/sby183)
• Chawner, S. et al. 2019. Genotype-phenotype relationships in children with copy number variants associated with high neuropsychiatric risk: Findings from the Intellectual Disability & Mental Health: Assessing the Genomic Impact on Neurodevelopment (IMAGINE-ID) study.. bioRxiv (10.1101/535708)
• Chawner, S. J. R. A. et al. 2019. Genotype–phenotype associations in children with copy number variants associated with high neuropsychiatric risk in the UK (IMAGINE-ID): a case-control cohort study. Lancet Psychiatry 6 (6), pp.493 - 505. (10.1016/S2215-0366(19)30123-3)
• Clifton, N. E. et al. 2019. Dynamic expression of genes associated with schizophrenia and bipolar disorder across development. Translational Psychiatry 9 74. (10.1038/s41398-019-0405-x)
• Crawford, K. et al. 2019. Medical consequences of pathogenic CNVs in adults: Analysis of the UK Biobank. Journal of Medical Genetics 56 , pp.131-138. (10.1136/jmedgenet-2018-105477)
• Cunningham, A. et al. 2019. Using kinematic analyses to explore sensorimotor control impairments in children with 22q11.2 deletion Syndrome. Journal of Neurodevelopmental Disorders 11 8. (10.1186/s11689-019-9271-3)
• Czamara, D. et al., 2019. Integrated analysis of environmental and genetic influences on cord blood DNA methylation in new-borns. Nature Communications 10 2548. (10.1038/s41467-019-10461-0)
• Drakesmith, M. et al. 2019. Genetic risk for schizophrenia and developmental delay is associated with shape and microstructure of midline white-matter structures. Translational Psychiatry 9 (1) 102. (10.1038/s41398-019-0440-7)
• Eaton, C. et al. 2019. Epilepsy and seizures in young people with 22q11.2 deletion syndrome: prevalence and links with other neurodevelopmental disorders. Epilepsia 60 (5), pp.818-829. (10.1111/epi.14722)
• Escott-Price, V. et al. 2019. Genetic liability to schizophrenia is negatively associated with educational attainment in UK Biobank. Journal of Molecular Psychiatry , pp.-. (10.1038/s41380-018-0328-6)
• Escott-Price, V. et al. 2019. Polygenic risk for schizophrenia and season of birth within the UK Biobank cohort. Psychological Medicine 49 (15), pp.2499-2504. (10.1017/S0033291718000454)
• Gorman, K. M. et al., 2019. Bi-allelic loss-of-function CACNA1B mutations in progressive epilepsy-dyskinesia. American Journal of Human Genetics 104 (5), pp.948-956. (10.1016/j.ajhg.2019.03.005)
• Harold, D. et al., 2019. Population-based identity-by-descent mapping combined with exome sequencing to detect rare risk variants for schizophrenia. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 180 (3), pp.223-231. (10.1002/ajmg.b.32716)
• Huckins, L. M. et al., 2019. Gene expression imputation across multiple brain regions provides insights into schizophrenia risk. Nature Genetics 51 , pp.659-674. (10.1038/s41588-019-0364-4)
• Kendall, K. M. et al. 2019. Cognitive performance and functional outcomes of carriers of pathogenic copy number variants: analysis of the UK Biobank. British Journal of Psychiatry 214 (05), pp.297-304. (10.1192/bjp.2018.301)
• Kendall, K. M. et al. 2019. Association of rare copy number variants with risk of depression. JAMA Psychiatry 76 (8), pp.818-825. (10.1001/jamapsychiatry.2019.0566)
• Kunkle, B. W. et al., 2019. Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing. Nature Genetics 51 (3), pp.414-430. (10.1038/s41588-019-0358-2)
• Lancaster, T. M. et al. 2019. Structural and functional neuroimaging of polygenic risk for schizophrenia: a recall-by-genotype-based approach. Schizophrenia Bulletin 45 (2), pp.405-414. (10.1093/schbul/sby037)
• Legge, S. E. et al. 2019. Association of genetic liability to psychotic experiences with neuropsychotic disorders and traits. JAMA Psychiatry 76 (12), pp.1256-1265. (10.1001/jamapsychiatry.2019.2508)
• Legge, S. E. et al. 2019. A genome-wide association study in individuals of African ancestry reveals the importance of the Duffy-null genotype in the assessment of clozapine-related neutropenia. Molecular Psychiatry 24 , pp.328-337. (10.1038/s41380-018-0335-7)
• Mulllins, N. et al., 2019. GWAS of suicide attempt in psychiatric disorders identifies association with major depression polygenic risk scores. American Journal of Psychiatry 176 (8), pp.651-660. (10.1176/appi.ajp.2019.18080957)
• Niarchou, M. et al. 2019. Genome-wide association study of dietary intake in the UK biobank study and its associations with schizophrenia and other traits. Translational Psychiatry 10 51. (10.1038/s41398-020-0688-y)
• Niarchou, M. et al. 2019. Attention deficit hyperactivity disorder symptoms as antecedents of later psychotic outcomes in 22q11.2 deletion syndrome. Schizophrenia Research 204 , pp.320-325. (10.1016/j.schres.2018.07.044)
• Niarchou, M. et al. 2019. Psychiatric disorders in children with 16p11.2 deletion and duplication. Translational Psychiatry (10.1038/s41398-018-0339-8)
• Pain, O. et al. 2019. Novel insight into the etiology of autism spectrum disorder gained by integrating expression data with genome-wide association statistics. Biological Psychiatry 86 (4), pp.265-273. (10.1016/j.biopsych.2019.04.034)
• Pardinas, A. F. et al. 2019. Pharmacogenomic variants and drug interactions identified through the genetic analysis of clozapine metabolism. American Journal of Psychiatry 176 (6), pp.477-486. (10.1176/appi.ajp.2019.18050589)
• Rees, E. et al. 2019. Targeted sequencing of 10,198 samples confirms abnormalities in neuronal activity and implicates voltage-gated sodium channels in schizophrenia pathogenesis. Biological Psychiatry 85 (7), pp.554-562. (10.1016/j.biopsych.2018.08.022)
• Richards, A. et al. 2019. Associations between schizophrenia genetic risk, anxiety disorders and manic/hypomanic episode in a New Zealand longitudinal population cohort study. British Journal of Psychiatry 214 (2), pp.96-102. (10.1192/bjp.2018.227)
• Romagnoni, A. et al., 2019. Comparative performances of machine learning methods for classifying Crohn Disease patients using genome-wide genotyping data. Scientific Reports 9 (1), pp.-. 10351. (10.1038/s41598-019-46649-z)
• Silva, A. I. et al., 2019. Cyfip1 haploinsufficient rats show white matter changes, myelin thinning, abnormal oligodendrocytes and behavioural inflexibility. Nature Communications 10 3455. (10.1038/s41467-019-11119-7)
• Silva, A. I. et al. 2019. Reciprocal white matter changes associated with copy number variation at 15q11.2 BP1-BP2: A diffusion tensor imaging study. Biological Psychiatry 85 (7), pp.563-572. (10.1016/j.biopsych.2018.11.004)
• Smeeth, D. M. et al., 2019. Polygenic risk for circulating reproductive hormone levels and their influence on hippocampal volume and depression susceptibility. Psychoneuroendocrinology 106 , pp.284-292. (10.1016/j.psyneuen.2019.04.011)
• Stahl, E. A. et al., 2019. Genome-wide association study identifies 30 loci associated with bipolar disorder. Nature Genetics 51 , pp.793-803. (10.1038/s41588-019-0397-8)
• Sykes, L. et al. 2019. Genetic variation in the psychiatric risk gene CACNA1C modulates reversal learning across species. Schizophrenia Bulletin 45 (5), pp.1024-1032. (10.1093/schbul/sby146)
• Vivian-Griffiths, T. et al. 2019. Predictive modeling of schizophrenia from genomic data: Comparison of polygenic risk score with kernel support vector machines approach. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 180 (1), pp.80-85. (10.1002/ajmg.b.32705)
• Zhang, X. et al., 2019. Genome-wide burden of rare short deletions is enriched in major depressive disorder in four cohorts. Biological Psychiatry 85 (12), pp.1065-1073. (10.1016/j.biopsych.2019.02.022)

2018

• Allardyce, J. et al. 2018. Association between schizophrenia-related polygenic liability and the occurrence and level of mood-incongruent psychotic symptoms in bipolar disorder. JAMA Psychiatry 75 (1), pp.28-35. (10.1001/jamapsychiatry.2017.3485)
• Anttila, V. et al., 2018. Analysis of shared heritability in common disorders of the brain. Science 360 (6395) eaap8757. (10.1126/science.aap8757)
• Arnau-Soler, A. et al., 2018. Genome-wide interaction study of a proxy for stress-sensitivity and its prediction of major depressive disorder. PLoS ONE 13 (12) e0209160. (10.1371/journal.pone.0209160)
• Brieva, J. et al., 2018. Alternative diffusion anisotropy measures for the investigation of white matter alterations in 22q11.2 deletion syndrome. Presented at: 14th International Symposium on Medical Information Processing and Analysis, Mazatlán, Mexico 24-26 October 2018. Published in: Romero, E. , Lepore, N. and Brieva, J. eds. 14th International Symposium on Medical Information Processing and Analysis. Vol. 10975.Proceedings of SPIE Bellingham, Washington: SPIE. , pp.51. (10.1117/12.2513788)
• Chawner, S. J. et al. 2018. The emergence of psychotic experiences in the early adolescence of 22q11.2 Deletion Syndrome. Journal of Psychiatric Research 109 , pp.10-17. (10.1016/j.jpsychires.2018.11.002)
• Corbin, L. et al., 2018. Formalising recall by genotype as an efficient approach to detailed phenotyping and causal inference. Nature Communications 9 711. (10.1038/s41467-018-03109-y)
• Cosgrove, D. et al., 2018. Effects of MiR-137 genetic risk score on brain volume and cortical measures in patients with schizophrenia and controls. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 177 (3), pp.369-376. (10.1002/ajmg.b.32620)
• Cunningham, A. C. et al. 2018. Developmental coordination disorder, psychopathology and IQ in 22q11.2 deletion syndrome. British Journal of Psychiatry 212 (1), pp.27-33. (10.1192/bjp.2017.6)
• Donohoe, G. et al., 2018. Genetically predicted complement component 4A expression: effects on memory function and middle temporal lobe activation. Psychological Medicine 48 (10), pp.1608-1615. (10.1017/S0033291717002987)
• Guyatt, A. L. et al., 2018. Association of copy number variation across the genome with neuropsychiatric traits in the general population. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 177 (5), pp.489-502. (10.1002/ajmg.b.32637)
• John, A. et al. 2018. Premature mortality among people with severe mental illness - new evidence from linked primary care data. Schizophrenia Research 199 , pp.154-162. (10.1016/j.schres.2018.04.009)
• Jones, H. J. et al., 2018. Investigating the genetic architecture of general and specific psychopathology in adolescence. Translational Psychiatry 8 (1) 145. (10.1038/s41398-018-0204-9)
• Leonenko, G. et al. 2018. A data-driven investigation of relationships between bipolar psychotic symptoms and schizophrenia genome-wide significant genetic loci. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 177 (4), pp.468-475. (10.1002/ajmg.b.32635)
• Li, M. et al., 2018. Integrative functional genomic analysis of human brain development and neuropsychiatric risks. Science 362 (6420) eaat7615. (10.1126/science.aat7615)
• Lynham, A. et al. 2018. Examining cognition across the bipolar / schizophrenia diagnostic spectrum. Journal of Psychiatry and Neuroscience 43 (3) 170076. (10.1503/jpn.170076)
• Morrison, S. et al. 2018. Vulnerable periods for cognitive development in individuals at high genomic risk of schizophrenia [Conference Abstract]. Schizophrenia Bulletin 44 (suppl), pp.S86. (10.1093/schbul/sby015.214)
• O'Brien, H. E. et al. 2018. Expression quantitative trait loci in the developing human brain and their enrichment in neuropsychiatric disorders. Genome Biology 19 194. (10.1186/s13059-018-1567-1)
• Owen, D. et al. 2018. Effects of pathogenic CNVs on physical traits in participants of the UK Biobank. BMC Genomics 19 (1) 867. (10.1186/s12864-018-5292-7)
• Pardinas, A. F. et al. 2018. Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection. Nature Genetics 50 , pp.381-389. (10.1038/s41588-018-0059-2)
• Ruderfer, D. M. et al., 2018. Genomic dissection of bipolar disorder and schizophrenia, including 28 subphenotypes. Cell 173 (7), pp.1705-1715.e16. (10.1016/j.cell.2018.05.046)
• Skene, N. G. et al., 2018. Genetic identification of brain cell types underlying schizophrenia. Nature Genetics 50 , pp.825-833. (10.1038/s41588-018-0129-5)
• Wray, N. R. et al., 2018. Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression. Nature Genetics 50 (5), pp.668-681. (10.1038/s41588-018-0090-3)
• Zhao, Y. et al., 2018. Variance of IQ is partially dependent on deletion type among 1,427 22q11.2 deletion syndrome subjects. American Journal of Medical Genetics Part A 176 (10), pp.2172-2181. (10.1002/ajmg.a.40359)

2017

• Allardyce, J. et al. 2017. Psychosis and the level of mood incongruence in bipolar disorder are related to genetic liability for schizophrenia. bioRxiv
• Bassett, A. S. et al., 2017. Rare genome-wide copy number variation and expression of schizophrenia in 22q11.2 deletion syndrome. American Journal of Psychiatry 174 (11), pp.1054-1063. (10.1176/appi.ajp.2017.16121417)
• Bigdeli, T. B. et al., 2017. Genetic effects influencing risk for major depressive disorder in China and Europe. Translational Psychiatry 7 (3), pp.e1074. (10.1038/tp.2016.292)
• Chawner, S. et al. 2017. Childhood cognitive development in 22q11.2 deletion syndrome: case–control study. British Journal of Psychiatry 211 (4), pp.223-230. (10.1192/bjp.bp.116.195651)
• Clifton, N. E. et al. 2017. Schizophrenia copy number variants and associative learning. Molecular Psychiatry 22 (2), pp.178-182. (10.1038/mp.2016.227)
• Cosgrove, D. et al., 2017. MiR-137-derived polygenic risk: effects on cognitive performance in patients with schizophrenia and controls. Translational Psychiatry 7 (1), pp.e1012. (10.1038/tp.2016.286)
• Cosgrove, D. et al., 2017. Cognitive characterization of schizophrenia risk variants involved in synaptic transmission: evidence of CACNA1C's role in working memory. Neuropsychopharmacology 42 , pp.2612-2622. (10.1038/npp.2017.123)
• Demaerel, W. et al., 2017. Nested inversion polymorphisms predispose chromosome 22q11.2 to meiotic rearrangements [RETRACTED]. American Journal of Human Genetics 101 (4), pp.616-622. (10.1016/j.ajhg.2017.09.002)
• Giegling, I. et al., 2017. Genetics of schizophrenia: A consensus paper of the WFSBP task force on genetics. World Journal of Biological Psychiatry 18 (7), pp.492-505. (10.1080/15622975.2016.1268715)
• Green, E. K. et al., 2017. Genome-wide significant locus for Research Diagnostic Criteria Schizoaffective Disorder Bipolar type. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 174 (8), pp.767-771. (10.1002/ajmg.b.32572)
• Hendricks, A. E. et al., 2017. Rare variant analysis of human and rodent obesity genes in individuals with severe childhood obesity. Scientific Reports 7 4394. (10.1038/s41598-017-03054-8)
• Hensman Moss, D. J. et al., 2017. Identification of genetic variants associated with Huntington's disease progression: a genome-wide association study. Lancet Neurology 16 (9), pp.701-711. (10.1016/S1474-4422(17)30161-8)
• Howes, O. D. et al., 2017. The role of genes, stress, and dopamine in the development of schizophrenia. Biological psychiatry 81 (1), pp.9-20. (10.1016/j.biopsych.2016.07.014)
• Kendall, K. , Kirov, G. and Owen, M. 2017. Schizophrenia Genetics. In: Benjamin, S. , Virginia, S. and Pedro, R. eds. Kaplan and Sadock?s Comprehensive Textbook of Psychiatry. Wolters Kluwer
• Kendall, K. M. et al. 2017. Cognitive performance among carriers of pathogenic copy number variants: analysis of 152,000 UK Biobank subjects. Biological Psychiatry 82 (2), pp.P103-110. (10.1016/j.biopsych.2016.08.014)
• Legge, S. E. et al. 2017. Genome-wide common and rare variant analysis provides novel insights into clozapine-associated neutropenia. Molecular Psychiatry 22 , pp.1502-1508. (10.1038/mp.2016.97)
• Leonenko, G. et al. 2017. Investigation of relationships between bipolar disorder phenotypes and genome-wide significant loci from PGC2 schizophrenia. European Neuropsychopharmacology 27 (S3), pp.S383-S384. (10.1016/j.euroneuro.2016.09.416)
• Leonenko, G. et al. 2017. Mutation intolerant genes and targets of FMRP are enriched for nonsynonymous alleles in schizophrenia. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 174 (7), pp.724-731. (10.1002/ajmg.b.32560)
• Marshall, C. R. et al., 2017. Contribution of copy number variants to schizophrenia from a genome-wide study of 41,321 subjects. Nature Genetics 49 , pp.27-35. (10.1038/ng.3725)
• McLaughlin, R. L. et al., 2017. Genetic correlation between amyotrophic lateral sclerosis and schizophrenia. Nature Communications 8 14774. (10.1038/ncomms14774)
• Peall, K. J. et al. 2017. A review of psychiatric co-morbidity described in genetic and immune mediated movement disorders. Neuroscience & Biobehavioral Reviews 80 , pp.23-35. (10.1016/j.neubiorev.2017.05.014)
• Rivera, M. et al., 2017. Interaction between the FTO gene, body mass index and depression: meta-analysis of 13701 individuals. British Journal of Psychiatry 211 (2), pp.70-76. (10.1192/bjp.bp.116.183475)
• Sims, R. et al. 2017. Rare coding variants in PLCG2, ABI3 and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease. Nature Genetics 49 , pp.1373-1384. (10.1038/ng.3916)
• Singh, T. et al., 2017. The contribution of rare variants to risk of schizophrenia in individuals with and without intellectual disability. Nature Genetics 49 , pp.1167-1173. (10.1038/ng.3903)
• Witt, S. H. et al., 2017. Genome-wide association study of borderline personality disorder reveals genetic overlap with the bipolar disorder, schizophrenia and major depression. Translational Psychiatry 7 e1155.

2016

• Carroll, L. S. et al., 2016. Mutation screening of SCN2A in schizophrenia and identification of a novel loss-of-function mutation. Psychiatric Genetics 26 (2), pp.60-65. (10.1097/YPG.0000000000000110)
• Collishaw, S. et al. 2016. Mental health resilience in the adolescent offspring of parents with depression: a prospective longitudinal study. Lancet Psychiatry 3 (1), pp.49-57. (10.1016/S2215-0366(15)00358-2)
• D'Angelo, D. et al., 2016. Defining the Effect of the 16p11.2 Duplication on Cognition, Behavior, and Medical Comorbidities. JAMA Psychiatry 73 (1), pp.20-30. (10.1001/jamapsychiatry.2015.2123)
• Franke, B. et al., 2016. Genetic influences on schizophrenia and subcortical brain volumes: large-scale proof of concept. Nature Neuroscience 19 (3), pp.420-431. (10.1038/nn.4228)
• Fry, A. E. et al. 2016. Pathogenic copy number variants and SCN1A mutations in patients with intellectual disability and childhood-onset epilepsy. BMC Medical Genetics 17 , pp.-. 34. (10.1186/s12881-016-0294-2)
• Han, J. et al. 2016. Gender differences in CNV burden do not confound schizophrenia CNV associations. Scientific Reports 6 25986. (10.1038/srep25986)
• Harrison, J. and Owen, M. J. 2016. Alzheimer's disease: the amyloid hypothesis on trial. British Journal of Psychiatry 208 (1), pp.1-3. (10.1192/bjp.bp.115.167569)
• Hubbard, L. et al. 2016. Evidence of common genetic overlap between schizophrenia and cognition. Schizophrenia Bulletin 42 (3), pp.832-842. (10.1093/schbul/sbv168)
• Isles, A. R. et al. 2016. Parental origin of interstitial duplications at 15q11.2-q13.3 in schizophrenia and neurodevelopmental disorders. PLoS Genetics 12 (5) e1005993. (10.1371/journal.pgen.1005993)
• Jones, H. J. et al., 2016. Phenotypic manifestation of genetic risk for schizophrenia during adolescence in the general population. JAMA Psychiatry 73 (3), pp.221-228. (10.1001/jamapsychiatry.2015.3058)
• Jun, G. et al., 2016. A novel Alzheimer disease locus located near the gene encoding tau protein. Molecular Psychiatry 21 , pp.108-117. (10.1038/mp.2015.23)
• Lancaster, T. et al. 2016. Associations between polygenic risk for schizophrenia and brain function during probabilistic learning in healthy individuals. Human Brain Mapping 37 (2), pp.491-500. (10.1002/hbm.23044)
• Legge, S. et al. 2016. Reasons for discontinuing clozapine: a cohort study of patients commencing treatment. Schizophrenia Research 174 , pp.113-119. (10.1016/j.schres.2016.05.002)
• Millan, M. J. et al., 2016. Altering the course of schizophrenia: progress and perspectives. Nature Reviews Drug Discovery 18 (7), pp.485-515. (10.1038/nrd.2016.28)
• Mooney, M. A. et al., 2016. Pathway analysis in attention deficit hyperactivity disorder: an ensemble approach. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 171 (6), pp.815-826. (10.1002/ajmg.b.32446)
• Mullins, N. et al., 2016. Polygenic interactions with environmental adversity in the aetiology of major depressive disorder. Psychological Medicine 46 (4), pp.759-770. (10.1017/S0033291715002172)
• O'Donovan, M. C. and Owen, M. J. 2016. The implications of the shared genetics of psychiatric disorders. Nature Medicine 22 , pp.1214-1219. (10.1038/nm.4196)
• Owen, M. J. and Doherty, J. L. 2016. What can we learn from the high rates of schizophrenia in people with 22q11.2 deletion syndrome?. World Psychiatry 15 (1), pp.23-25. (10.1002/wps.20274)
• Owen, M. J. , Sawa, A. and Mortensen, P. B. 2016. Schizophrenia. The Lancet 388 (10039), pp.86-97. (10.1016/S0140-6736(15)01121-6)
• Pardinas, A. et al. 2016. Common schizophrenia alleles are enriched in mutation-intolerant genes and maintained by background selection. bioRxiv (10.1101/068593)
• Peall, K. J. et al. 2016. Psychiatric disorders, myoclonus dystonia and SGCE: an international study. Annals of Clinical and Translational Neurology 3 (1), pp.4-11. (10.1002/acn3.263)
• Rees, E. et al. 2016. Analysis of intellectual disability copy number variants for association with schizophrenia. JAMA Psychiatry 73 (9), pp.963-969. (10.1001/jamapsychiatry.2016.1831)
• Richards, A. et al. 2016. Exome arrays capture polygenic rare variant contributions to schizophrenia. Human Molecular Genetics 25 (5), pp.1001-1007. (10.1093/hmg/ddv620)
• Rucker, J. J. et al., 2016. Phenotypic association analyses with copy number variation in recurrent depressive disorder. Biological Psychiatry 79 (4), pp.329-336. (10.1016/j.biopsych.2015.02.025)
• Singh, T. et al., 2016. Rare loss-of-function variants in KMT2F are associated with schizophrenia and developmental disorders. Nature Neuroscience 19 , pp.571-577. (10.1101/036384)
• Singh, T. et al., 2016. Rare loss-of-function variants in SETD1A are associated with schizophrenia and developmental disorders. Nature Neuroscience 19 (4), pp.571-577. (10.1038/nn.4267)
• Stephan, K. E. et al., 2016. Charting the landscape of priority problems in psychiatry, part 1: classification and diagnosis. The Lancet Psychiatry 3 (1), pp.77-83. (10.1016/S2215-0366(15)00361-2)
• Stephan, K. E. et al., 2016. Charting the landscape of priority problems in psychiatry, part 2: pathogenesis and aetiology. The Lancet Psychiatry 3 (1), pp.84-90. (10.1016/S2215-0366(15)00360-0)
• Tansey, K. E. et al. 2016. Common alleles contribute to schizophrenia in CNV carriers. Molecular Psychiatry 21 , pp.1085-1089. (10.1038/mp.2015.143)
• Thapar, A. et al. 2016. Psychiatric gene discoveries shape evidence on ADHD's biology. Molecular Psychiatry 21 , pp.1202-1207. (10.1038/mp.2015.163)
• Walters, J. T. R. and Owen, M. J. 2016. Genome-wide significant associations for cannabis dependence severity. JAMA Psychiatry 73 (5), pp.443-444. (10.1001/jamapsychiatry.2016.0046)
• Whitton, L. et al., 2016. Cognitive analysis of schizophrenia risk genes that function as epigenetic regulators of gene expression. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 171 (8), pp.1170-1179. (10.1002/ajmg.b.32503)

2015

• Desikan, R. S. et al., 2015. Genetic overlap between Alzheimer's Disease and Parkinson's Disease at the MAPT locus. Molecular Psychiatry 20 (12), pp.1588-1595. (10.1038/mp.2015.6)
• Escott-Price, V. et al. 2015. No evidence for enrichment in schizophrenia for common allelic associations at imprinted loci. PLoS ONE 10 (12), pp.-. e0144172. (10.1371/journal.pone.0144172)
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2014

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2013

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2012

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• Owen, M. J. 2012. Intellectual disability and major psychiatric disorders: a continuum of neurodevelopmental causality. British Journal of Psychiatry 200 (4), pp.268-269. (10.1192/bjp.bp.111.105551)
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• Peall, K. J. et al. 2012. Myoclonus dystonia syndrome: SGCE mutations and psychiatric disease [Abstract]. Journal of Neurology 259 , pp.S30-S30.
• Power, R. A. et al., 2012. Dissecting the genetic heterogeneity of depression through age at onset. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 159B (7), pp.859-868. (10.1002/ajmg.b.32093)
• Proitsi, P. et al., 2012. Association of serotonin and dopamine gene pathways with behavioral subphenotypes in dementia. Neurobiology of Aging 33 (4), pp.791-803. (10.1016/j.neurobiolaging.2010.06.011)
• Rees, E. et al. 2012. De Novo mutation in schizophrenia. Schizophrenia Bulletin 38 (3), pp.377-381. (10.1093/schbul/sbs047)
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• Sellers, R. et al. 2012. Risk of psychopathology in adolescent offspring of mothers with psychopathology and recurrent depression. British Journal of Psychiatry 202 (2), pp.108-114. (10.1192/bjp.bp.111.104984)
• Shi, H. et al., 2012. Genetic variants influencing human aging from late-onset Alzheimer's disease (LOAD) genome-wide association studies (GWAS). Neurobiology of Aging 33 (8) 1849.e5. (10.1016/j.neurobiolaging.2012.02.014)
• Stergiakouli, E. et al. 2012. Investigating the contribution of common genetic variants to the risk and pathogenesis of ADHD. American Journal of Psychiatry 169 (2), pp.186-194. (10.1176/appi.ajp.2011.11040551)
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• Thapar, A. K. et al. 2012. Trajectories of change in self-reported psychotic-like experiences in childhood and adolescence. Schizophrenia Research 140 (1-3), pp.104-109. (10.1016/j.schres.2012.06.024)
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• Williams, N. M. et al. 2012. Genome-wide analysis of copy number variants in attention deficit hyperactivity disorder: the role of rare variants and duplications at 15q13.3. American Journal of Psychiatry 169 (2), pp.195-204. (10.1176/appi.ajp.2011.11060822)

2011

• Belbin, O. et al., 2011. A multi-center study of ACE and the risk of late-onset Alzheimer's disease. Journal of Alzheimer's Disease 24 (3), pp.587-597. (10.3233/JAD-2011-101914)
• Boot, E. et al., 2011. COMT Val158met genotype and striatal D2/3 receptor binding in adults with 22q11 deletion syndrome. Synapse 65 (9), pp.967-970. (10.1002/syn.20932)
• Breen, G. et al., 2011. A genome-wide significant linkage for severe depression on chromosome 3: the depression network study. American Journal of Psychiatry 168 (8), pp.840-847. (10.1176/appi.ajp.2011.10091342)
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• Cardno, A. G. et al., 2011. A genomewide linkage study of age at onset in schizophrenia. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 156B (8), pp.929-940. (10.1002/ajmg.1404)
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• Chen, J. et al., 2011. Two non-synonymous markers in PTPN21, identified by genome-wide association study data-mining and replication, are associated with schizophrenia. Schizophrenia Research 131 (1-3), pp.43-51.
• Chen, J. et al., 2011. Two non-synonymous markers in PTPN21, identified by genome-wide association study data-mining and replication, are associated with schizophrenia. Schizophrenia Research 131 (1-3), pp.43-51. (10.1016/j.schres.2011.06.023)
• Cichon, S. et al., 2011. Genome-wide association study identifies genetic variation in neurocan as a susceptibility factor for bipolar disorder. American Journal of Human Genetics 88 (3), pp.372-381. (10.1016/j.ajhg.2011.01.017)
• Donohoe, G. et al., 2011. A neuropsychological investigation of the genome wide associated schizophrenia risk variant NRGN rs12807809 [Letter]. Schizophrenia Research 125 (2-3), pp.304-306. (10.1016/j.schres.2010.10.019)
• Dwyer, S. L. et al. 2011. Investigation of rare non-synonymous variants at ABCA13 in schizophrenia and bipolar disorder [letter]. Molecular Psychiatry 16 (8), pp.790-791. (10.1038/mp.2011.2)
• Escott-Price, V. et al. 2011. An examination of single nucleotide polymorphism selection prioritization strategies for tests of gene-gene interaction. Biological Psychiatry 70 (2), pp.198-203. (10.1016/j.biopsych.2011.01.034)
• Green, E. K. et al. 2011. DISC1 exon 11 rare variants found more commonly in schizoaffective spectrum cases than controls. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 156B (4), pp.490-492. (10.1002/ajmg.b.31187)
• Hamshere, M. L. et al. 2011. Phenotype evaluation and genomewide linkage study of clinical variables in schizophrenia. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 156 (8), pp.929-940. (10.1002/ajmg.b.31240)
• Hamshere, M. L. et al. 2011. Polygenic dissection of the bipolar phenotype. British Journal of Psychiatry 198 (4), pp.284-288. (10.1192/bjp.bp.110.087866)
• Hansen, T. et al., 2011. At-risk variant in TCF7L2 for type II diabetes increases risk of schizophrenia. Biological Psychiatry 70 (1), pp.59-63. (10.1016/j.biopsych.2011.01.031)
• Hinney, A. et al., 2011. Genome-wide association study in German patients with attention deficit/hyperactivity disorder. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 156 (8), pp.888-897. (10.1002/ajmg.b.31246)
• Hollingworth, P. et al. 2011. Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease. Nature Genetics 43 (5), pp.429-435. (10.1038/ng.803)
• Ikeda, M. et al., 2011. Genome-Wide Association Study of Schizophrenia in a Japanese Population. Biological Psychiatry 69 (5), pp.472-478. (10.1016/j.biopsych.2010.07.010)
• Ingason, A. et al., 2011. Maternally derived microduplications at 15q11-q13: Implication of imprinted genes in psychotic illness. American Journal of Psychiatry 168 (4), pp.408-417. (10.1176/appi.ajp.2010.09111660)
• Langley, K. et al. 2011. Clinical and cognitive characteristics of children with attention-deficit hyperactivity disorder, with and without copy number variants. British Journal of Psychiatry 199 (5), pp.398-403.
• O'Dushlaine, C. et al., 2011. Molecular pathways involved in neuronal cell adhesion and membrane scaffolding contribute to schizophrenia and bipolar disorder susceptibility. Molecular Psychiatry 16 (3), pp.286-292. (10.1038/mp.2010.7)
• Owen, M. J. 2011. Is there a schizophrenia to diagnose? [Editorial]. World Psychiatry 10 (1), pp.34-35. (10.1002/j.2051-5545.2011.tb00007.x)
• Owen, M. J. et al. 2011. Neurodevelopmental hypothesis of schizophrenia. British Journal of Psychiatry 198 (3), pp.173-175. (10.1192/bjp.bp.110.084384)
• Peall, K. J. et al. 2011. Psychiatric disorders, myoclonus dystonia, and the epsilon-sarcoglycan gene: a systematic review. Movement Disorders 26 (10), pp.1939-1942. (10.1002/mds.23791)
• Proitsi, P. et al., 2011. A Multiple Indicators Multiple Causes (MIMIC) model of Behavioural and Psychological Symptoms in Dementia (BPSD). Neurobiology of Aging 32 (3), pp.434-442. (10.1016/j.neurobiolaging.2009.03.005)
• Rees, E. et al. 2011. De novo rates and selection of schizophrenia-associated copy number variants. Biological Psychiatry 70 (12), pp.1109-1114. (10.1016/j.biopsych.2011.07.011)
• Reilly, M. P. et al., 2011. Identification of ADAMTS7 as a novel locus for coronary atherosclerosis and association of ABO with myocardial infarction in the presence of coronary atherosclerosis: two genome-wide association studies. The Lancet 377 (9763), pp.383-392. (10.1016/S0140-6736(10)61996-4)
• Reitz, C. et al., 2011. Meta-analysis of the association between variants in SORL1 and Alzheimer Disease. Archives of Neurology 68 (1), pp.99-106. (10.1001/archneurol.2010.346)
• Rietschel, M. et al., 2011. Association between genetic variation in a region on chromosome 11 and schizophrenia in large samples from Europe. Molecular Psychiatry , pp.1-12. (10.1038/mp.2011.80)
• Ripke, S. et al., 2011. Genome-wide association study identifies five new schizophrenia loci [Letter]. Nature Genetics 43 (10), pp.969-976. (10.1038/ng.940)
• Rivera, M. et al., 2011. Depressive disorder moderates the effect of the FTO gene on body mass index. Molecular Psychiatry 17 (6), pp.604-611. (10.1038/mp.2011.45)
• Ruderfer, D. M. et al., 2011. A family-based study of common polygenic variation and risk of schizophrenia [Letter]. Molecular Psychiatry 16 (9), pp.887-888. (10.1038/mp.2011.34)
• Samaan, Z. et al., 2011. Methylenetetrahydrofolate reductase gene variant (MTHFR C677T) and migraine: a case control study and meta-analysis. BMC Neurology 11 , pp.66-74. (10.1186/1471-2377-11-66)
• Schosser, A. et al., 2011. A follow-up case-control association study of tractable (druggable) genes in recurrent major depression. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 156 (6), pp.640-650. (10.1002/ajmg.b.31204)
• Schosser, A. et al., 2011. P02-217 - COMT in major depression - UK candidate gene association study [Meeting Abstract]. European Psychiatry 26 , pp.813. (10.1016/S0924-9338(11)72518-4)
• Schosser, A. et al., 2011. Genomewide association scan of suicidal thoughts and behaviour in major depression. PLoS ONE 6 (7) e20690. (10.1371/journal.pone.0020690)
• Sims, R. et al. 2011. No evidence that extended tracts of homozygosity are associated with Alzheimer's disease. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 156 (7), pp.764-771. (10.1002/ajmg.b.31216)
• Sklar, P. et al., 2011. Large-scale genome-wide association analysis of bipolar disorder identifies a new susceptibility locus near ODZ4 [Letter]. Nature Genetics 43 (10), pp.977-983. (10.1038/ng.943)
• Smith, R. L. et al. 2011. Analysis of neurogranin (NRGN) in schizophrenia. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 156B (5), pp.532-535. (10.1002/ajmg.b.31191)
• Steinberg, S. et al., 2011. Common variants at VRK2 and TCF4 conferring risk of schizophrenia. Human Molecular Genetics 20 (20), pp.4076-4081. (10.1093/hmg/ddr325)
• Stergiakouli, E. et al., 2011. Steroid sulfatase is a potential modifier of cognition in attention deficit hyperactivity disorder. Genes Brain and Behavior 10 (3), pp.334-344. (10.1111/j.1601-183x.2010.00672.x)
• Williams, H. J. et al. 2011. Most genome-wide significant susceptibility loci for schizophrenia and bipolar disorder reported to date cross-traditional diagnostic boundaries. Human Molecular Genetics 20 (2), pp.387-391. (10.1093/hmg/ddq471)
• Williams, H. J. et al. 2011. Association between TCF4 and schizophrenia does not exert its effect by common nonsynonymous variation or by influencing cis-acting regulation of mRNA expression in adult human brain. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 156 (7), pp.781-784. (10.1002/ajmg.b.31219)
• Williams, H. J. et al. 2011. Fine mapping of ZNF804A and genome-wide significant evidence for its involvement in schizophrenia and bipolar disorder. Molecular Psychiatry 16 (4), pp.429-441. (10.1038/mp.2010.36)
• Williams, N. M. et al. 2011. Structural variations in attention-deficit hyperactivity disorder - Authors' reply [Letter]. The Lancet 377 (9763), pp.378. (10.1016/S0140-6736(11)60121-9)
• Yosifova, A. et al., 2011. Genome-wide association study on bipolar disorder in the Bulgarian population. Genes, Brain and Behavior 10 (7), pp.789-797. (10.1111/j.1601-183X.2011.00721.x)
• Zammit, S. et al. 2011. Cannabis, COMT and psychotic experiences. British Journal of Psychiatry 199 (5), pp.380-385. (10.1192/bjp.bp.111.091421)
• Zuliani, R. et al., 2011. Genetic variants in the ErbB4 gene are associated with white matter integrity. Psychiatry Research: Neuroimaging 191 (2), pp.133-137. (10.1016/j.pscychresns.2010.11.001)

2010

• Allison, D. B. et al., 2010. Accurately assessing the risk of schizophrenia conferred by rare copy-number variation affecting genes with brain function. PLoS Genetics 6 (9) e1001097. (10.1371/journal.pgen.1001097)
• Baig, S. et al., 2010. Distribution and expression of picalm in alzheimer disease. Journal of Neuropathology and Experimental Neurology 69 (10), pp.1071-1077. (10.1097/NEN.0b013e3181f52e01)
• Blake, D. J. et al., 2010. TCF4, Schizophrenia, and Pitt-Hopkins Syndrome. Schizophrenia Bulletin 36 (3), pp.443-447. (10.1093/schbul/sbq035)
• Butler, A. W. et al., 2010. A genomewide linkage study on suicidality in major depressive disorder confirms evidence for linkage to 2p12. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 153B (8), pp.1465-1473. (10.1002/ajmg.b.31127)
• Carroll, L. S. et al. 2010. Evidence for rare and common genetic risk variants for schizophrenia at protein kinase C, alpha. Molecular Psychiatry 15 (11), pp.1101-1111. (10.1038/mp.2009.96)
• Chen, X. et al., 2010. GWA study data mining and independent replication identify cardiomyopathy-associated 5 (CMYA5) as a risk gene for schizophrenia. Molecular Psychiatry 16 (11), pp.1117-1129. (10.1038/mp.2010.96)
• Cohen-Woods, S. et al., 2010. The Bipolar Association Case-Control Study (BACCS) and meta-analysis: No association with the 5,10-Methylenetetrahydrofolate reductase gene and bipolar disorder. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 153B (7), pp.1298-1304. (10.1002/ajmg.b.31101)
• Craddock, N. J. et al. 2010. Genome-wide association study of CNVs in 16,000 cases of eight common diseases and 3,000 shared controls. Nature 464 (7289), pp.713-720. (10.1038/nature08979)
• Craddock, N. J. et al. 2010. Strong genetic evidence for a selective influence of GABAA receptors on a component of the bipolar disorder phenotype. Molecular Psychiatry 15 (2), pp.146-153. (10.1038/mp.2008.66)
• Craddock, N. J. et al. 2010. Strong genetic evidence for a selective influence of GABAA receptors on a component of the bipolar disorder phenotype [Corrigendum]. Molecular Psychiatry 15 (11), pp.1121-1121. (10.1038/mp.2010.62)
• Craddock, N. J. and Owen, M. J. 2010. Authors' reply:. British Journal of Psychiatry 196 (6), pp.495-496. (10.1192/bjp.196.6.495a)
• Craddock, N. J. and Owen, M. J. 2010. Data and clinical utility should be the drivers of changes to psychiatric classification [Letter]. British Journal of Psychiatry 197 (2), pp.158. (10.1192/bjp.197.2.158)
• Craddock, N. J. and Owen, M. J. 2010. Molecular genetics and the kraepelinian dichotomy: one disorder, two disorders, or do we need to start thinking afresh?. Psychiatric Annals 40 (2), pp.88-91. (10.3928/00485718-20100127-04)
• Craddock, N. J. and Owen, M. J. 2010. Molecular genetics and the relationship between epilepsy and psychosis [Letter]. British Journal of Psychiatry 197 (1), pp.75-76. (10.1192/bjp.197.1.75a)
• Craddock, N. J. and Owen, M. J. 2010. The Kraepelinian dichotomy - going, going... but still not gone (Reappraisal). British Journal of Psychiatry 196 (2), pp.92-95. (10.1192/bjp.bp.109.073429)
• Cruchaga, C. et al., 2010. SNPS associated with cerebrospinal fluid phospho-tau levels influence rate of decline in Alzheimer's disease. PLoS Genetics 6 (9) e1001101. (10.1371/journal.pgen.1001101)
• Dwyer, S. L. et al. 2010. No evidence that rare coding variants in ZNF804A confer risk of schizophrenia. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 153B (8), pp.1411-1416. (10.1002/ajmg.b.31117)
• Escott-Price, V. et al. 2010. Genetic differences between five European populations. Human Heredity 70 (2), pp.141-149. (10.1159/000313854)
• Green, E. K. et al. 2010. Variation at the GABAA receptor gene, Rho 1 (GABRR1) associated with susceptibility to bipolar schizoaffective disorder. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 153B (7), pp.1347-1349. (10.1002/ajmg.b.31108)
• Green, E. K. et al. 2010. The bipolar disorder risk allele at CACNA1C also confers risk of recurrent major depression and of schizophrenia. Molecular Psychiatry 15 (10), pp.1016-1022. (10.1038/mp.2009.49)
• Grozeva, D. et al. 2010. Rare copy number variants: A point of rarity in genetic risk for bipolar disorder and schizophrenia. Archives of General Psychiatry 67 (4), pp.318-327. (10.1001/archgenpsychiatry.2010.25)
• Hollingworth, P. et al. 2010. Alzheimer's disease genetics: current knowledge and future challenges. International Journal of Geriatric Psychiatry 26 (8), pp.793-802. (10.1002/gps.2628)
• Ikeda, M. et al. 2010. Copy number variation in schizophrenia in the Japanese population. Biological Psychiatry 67 (3), pp.283-286. (10.1016/j.biopsych.2009.08.034)
• Ikeda, M. et al. 2010. Identification of novel candidate genes for treatment response to risperidone and susceptibility for schizophrenia: integrated analysis among pharmacogenomics, mouse expression, and genetic case-control association approaches. Biological psychiatry 67 (3), pp.263-269. (10.1016/j.biopsych.2009.08.030)
• Ikeda, M. et al. 2010. Failure to confirm association between PIK4CA and psychosis in 22q11.2 deletion syndrome. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 153B (4), pp.980-982. (10.1002/ajmg.b.31060)
• Jones, L. et al. 2010. Genetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's disease. PLoS ONE 5 (11) e13950. (10.1371/journal.pone.0013950)
• Langley, K. et al. 2010. Adolescent clinical outcomes for young people with attention-deficit hyperactivity disorder. British Journal of Psychiatry 196 (3), pp.235-240. (10.1192/bjp.bp.109.066274)
• Langley, K. et al. 2010. Genotype link with extreme antisocial behavior: The contribution of cognitive pathways. Archives of General Psychiatry 67 (12), pp.1317-1323. (10.1001/archgenpsychiatry.2010.163)
• Lewis, C. M. et al., 2010. Genome-wide association study of major recurrent depression in the U.K. population. American Journal of Psychiatry 167 (8), pp.949-957. (10.1176/appi.ajp.2010.09091380)
• Liu, Y. et al., 2010. Meta-analysis of genome-wide association data of bipolar disorder and major depressive disorder [Letter]. Molecular Psychiatry 16 (1), pp.2-4. (10.1038/mp.2009.107)
• Lupton, M. K. et al., 2010. Education, occupation and retirement age effects on the age of onset of Alzheimer's disease. International Journal of Geriatric Psychiatry 25 (1), pp.30-36. (10.1002/gps.2294)
• Neale, B. M. et al., 2010. Meta-analysis of genome-wide association studies of attention-deficit/hyperactivity disorder. Journal of the American Academy of Child & Adolescent Psychiatry 49 (9), pp.884-897. (10.1016/j.jaac.2010.06.008)
• Nishikawa, K. et al., 2010. Maf promotes osteoblast differentiation in mice by mediating the age-related switch in mesenchymal cell differentiation. The Journal of Clinical Investigation 120 (10), pp.3455-3465. (10.1172/JCI42528)
• Owen, M. J. , Craddock, N. J. and O'Donovan, M. C. 2010. Suggestion of roles for both common and rare risk variants in genome-wide studies of schizophrenia. Archives of General Psychiatry 67 (7), pp.667-673. (10.1001/archgenpsychiatry.2010.69)
• Schosser, A. et al., 2010. Genome wide association scan of co-morbid anxiety in major depressive disorder (MDD) [Abstract]. International Journal of Psychiatry in Clinical Practice 14 (S1), pp.37.
• Seshadri, S. et al., 2010. Genome-wide analysis of genetic loci associated with Alzheimer Disease. JAMA - The Journal of the American Medical Association 303 (18), pp.1832-1840. (10.1001/jama.2010.574)
• Talkowski, M. E. et al., 2010. Fine-mapping reveals novel alternative splicing of the dopamine transporter. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 153B (8), pp.1434-1447. (10.1002/ajmg.b.31125)
• Walters, J. T. R. et al. 2010. Psychosis susceptibility gene ZNF804A and cognitive performance in schizophrenia. Archives of General Psychiatry 67 (7), pp.692-700. (10.1001/archgenpsychiatry.2010.81)
• Williams, N. M. et al. 2010. Rare chromosomal deletions and duplications in attention-deficit hyperactivity disorder: a genome-wide analysis. The Lancet 376 (9750), pp.1401-1408. (10.1016/S0140-6736(10)61109-9)
• Zammit, S. , Owen, M. J. and Lewis, G. 2010. Misconceptions about gene-environment interactions in psychiatry. Evidence Based Mental Health 13 (3), pp.65-68. (10.1136/ebmh.13.3.65)

2009

• Abraham, R. A. et al. 2009. An association study of common variation at theMAPTlocus with late-onset Alzheimer's disease. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 150B (8), pp.1152-1155. (10.1002/ajmg.b.30951)
• Azuma, R. et al., 2009. Visuospatial working memory in children and adolescents with 22q11.2 deletion syndrome; an fMRI study. Journal of Neurodevelopmental Disorders 1 (1), pp.46-60. (10.1007/s11689-009-9008-9)
• Ball, H. A. et al., 2009. Depression, migraine with aura and migraine without aura: their familiality and interrelatedness. Cephalagia 29 (8), pp.848-854. (10.1111/j.1468-2982.2008.01808.x)
• Betcheva, E. T. et al., 2009. Case-control association study of 59 candidate genes reveals the DRD2 SNP rs6277 (C957T) as the only susceptibility factor for schizophrenia in the Bulgarian population. Journal of Human Genetics 54 (2), pp.98-107. (10.1038/jhg.2008.14)
• Carroll, L. S. and Owen, M. J. 2009. Genetic overlap between autism, schizophrenia and bipolar disorder [Review]. Genome Medicine 1 (10), pp.102. (10.1186/gm102)
• Carroll, L. S. et al. 2009. Evidence that putative ADHD low risk alleles atSNAP25may increase the risk of schizophrenia. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 150B (7), pp.893-899. (10.1002/ajmg.b.30915)
• Cohen-Woods, S. et al., 2009. Depression Case Control (DeCC) Study fails to support involvement of the muscarinic acetylcholine receptor M2 (CHRM2) gene in recurrent major depressive disorder. Human Molecular Genetics 18 (8), pp.1504-1509. (10.1093/hmg/ddp051)
• Craddock, N. J. , O'Donovan, M. C. and Owen, M. J. 2009. Psychosis genetics: modeling the relationship between Schizophrenia, Bipolar Disorder, and Mixed (or "Schizoaffective") Psychoses. Schizophrenia Bulletin 35 (3), pp.482-490. (10.1093/schbul/sbp020)
• Donohoe, G. et al., 2009. Influence of NOS1 on verbal intelligence and working memory in both patients with schizophrenia and healthy control subjects. Archives of General Psychiatry 66 (10), pp.1045-1054. (10.1001/archgenpsychiatry.2009.139)
• Dwyer, S. L. et al. 2009. Mutation screening of the DTNBP1 exonic sequence in 669 schizophrenics and 710 controls using high-resolution melting analysis. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 153B (3), pp.766-774. (10.1002/ajmg.b.31045)
• Escott-Price, V. et al. 2009. Gene-wide analyses of genome-wide association data sets: evidence for multiple common risk alleles for schizophrenia and bipolar disorder and for overlap in genetic risk. Molecular Psychiatry 14 (3), pp.252-260. (10.1038/mp.2008.133)
• Fowler, T. A. et al. 2009. Psychopathy trait scores in adolescents with childhood ADHD: the contribution of genotypes affecting MAOA, 5HTT and COMT activity. Psychiatric Genetics 19 (6), pp.312-319. (10.1097/YPG.0b013e3283328df4)
• Fowler, T. A. et al. 2009. Psychopathy traits in adolescents with childhood attention-deficit hyperactivity disorder. British Journal of Psychiatry 194 (1), pp.62-67. (10.1192/bjp.bp.107.046870)
• Gerrish, A. et al. 2009. An examination of MUTED as a schizophrenia susceptibility gene [Letter]. Schizophrenia Research 107 (1), pp.110-111. (10.1016/j.schres.2008.08.011)
• Green, E. K. et al. 2009. P2RX7: A bipolar and unipolar disorder candidate susceptibility gene?. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 150B (8), pp.1063-1069. (10.1002/ajmg.b.30931)
• Hamshere, M. L. et al. 2009. Genetic utility of broadly defined bipolar schizoaffective disorder as a diagnostic concept. British Journal of Psychiatry 195 (1), pp.23-29. (10.1192/bjp.bp.108.061424)
• Hamshere, M. L. et al. 2009. Mood-incongruent psychosis in bipolar disorder: conditional linkage analysis shows genome-wide suggestive linkage at 1q32.3, 7p13 and 20q13.31. Bipolar Disorders 11 (6), pp.610-620. (10.1111/j.1399-5618.2009.00736.x)
• Harold, D. et al. 2009. Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease [Corrigendum]. Nature Genetics 41 (10), pp.1156. (10.1038/ng1009-1156d)
• Harold, D. et al. 2009. Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease [Letter]. Nature Genetics 41 (10), pp.1088-1093. (10.1038/ng.440)
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2008

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2007

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2006

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2005

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2004

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• Peirce, T. R. et al., 2004. Convergent functional genomics, association and linkage analysis suggests 2 ',3 '-cyclic nucleotide 3 '-phosphodiesterase (CNP) as a potential susceptibility gene for schizophrenia [Conference Abstract]. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics 130B (1), pp.81-81. (10.1002/ajmg.b.30101)
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2003

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2002

• Anney, R. et al. 2002. Characterisation, mutation detection, and association analysis of alternative promoters and 5' UTRs of the human dopamine D3 receptor gene in schizophrenia. Molecular psychiatry 7 (5), pp.493-502. (10.1038/sj.mp.4001003)
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2001

• 2001. Evidence of association between DRD4 and ADHD with conduct disturbance. Presented at: Behavior Genetics Association Meeting Cambridge 11 July 2001. Published in: Thapar, A. et al. Behavior Genetics. Vol. 31.Behavior Genetics Association Meeting Abstracts Vol. 5. Springer Verlag. , pp.470-471.
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• Kirov, G. , Stehens, M. and Owen, M. J. 2001. Screening of three candidate genes in the bipolar candidate region on chromosome 21q22.3. American Journal of Medical Genetics 105 (7), pp.612-612.
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• Rees, M. I. et al., 2001. A missense mutation of human Gephyrin (GPHN) is associated with Hyperekplexia and transcript isoform analysis re-defines the genomic structure of GPHN. American Journal of Human Genetics 69 (4), pp.627-627.
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2000

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• Elvidge, G. P. , Owen, M. J. and Craddock, N. J. 2000. Mutational analysis of purinergic receptor, P2X4: A positional candidate susceptibility gene for bipolar disorder.. American Journal of Medical Genetics 96 (4), pp.493-493.
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• Jacobsen, N. J. et al., 2000. Genomic characterisation and mutational analysis of CUX2, a potential regulator of NCAM expression, mapping to chromosome 12q23-q24.1.. American Journal of Medical Genetics 96 (4), pp.493-494.
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• Jonsson, E. G. et al., 2000. A promoter polymorphism in the monoamine oxidase A gene and its relationships to monoamine metabolite concentrations in CSF of healthy volunteers. Journal of Psychiatric Research 34 (3), pp.239-244. (10.1016/S0022-3956(00)00013-3)
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• Kirov, G. et al. 2000. Automated genotyping of single-nucleotide polymorphisms by extension of fluorescently labelled primers: analysis of individual and pooled DNA samples. Balkan Journal of Medical Genetics 3 , pp.23-28.
• Kirov, G. et al. 2000. Pooled genotyping of microsatellite markers in parent-offspring trios. Genome Research 10 (1), pp.105-115.
• Levinson, D. F. et al., 2000. Multicenter linkage study of schizophrenia candidate regions on chromosomes 5q, 6q, 10p, and 13q: schizophrenia linkage collaborative group III. American Journal of Human Genetics 67 (3), pp.652-663. (10.1086/303041)
• McCarthy, G. M. et al., 2000. Investigating familiality of subtypes in schizophrenia. American Journal of Medical Genetics 96 (4), pp.473-474.
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• Murphy, K. C. and Owen, M. J. 2000. Evidence for association between polymorphisms of the catechol-O-methyltransferase (COMT) and monoamine oxidase (MAO) genes and schizophrenia in adults with velo-cardio-facial syndrome.. American Journal of Medical Genetics 96 (4), pp.476-476.
• Murphy, K. C. et al., 2000. Chromosome 22q, velo-cardio-facial syndrome (VCFS) and schizophrenia. Journal of Medical Genetics 37 , pp.S58-S58.
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• Murphy, K. and Owen, M. J. 2000. No association between polymorphisms of catechol-o-methyltransferase (COMT) or monoamine oxidase (MAO) genes and schizophrenia in adults with velo-cardio-facial syndrome. Schizophrenia Research 41 (1), pp.96. (10.1016/S0920-9964(00)90531-0)
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• Payton, A. et al., 2000. A family based candidate gene association study of Attention Deficit Hyperactivity Disorder. American Journal of Medical Genetics 96 (4), pp.489-489.
• Rees, M. I. et al., 2000. A transient hyperekplexia phenotype associated with compound heterozygote mutations in the human beta-subunit of the inhibitory glycine receptor (GLRB). American Journal of Human Genetics 67 (4), pp.391-391.
• Rees, M. I. et al., 2000. Further evidence of autosomal dominant congenital zonular pulverulent cataracts linked to 13q11 (CZP3) and a novel mutation in connexin 46 (GJA3).. Human Genetics 106 (2), pp.206-209. (10.1007/s004390051029)
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1999

• Amelsvoort, T. V. et al., 1999. An MRI and fMRI study on the effect of deletions on chromosome 22 on brain. Biological psychiatry 45 (8), pp.110S-111S.
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• Hill, L. et al., 1999. IGF2R and cognitive ability. Molecular Psychiatry 4 , pp.S108-S108.
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• Hoogendoorn, B. et al. 1999. Genotyping single nucleotide polymorphisms by primer extension and high performance liquid chromatography. Human Genetics 104 (1), pp.89-93. (10.1007/s004390050915)
• Jacobsen, N. J. et al., 1999. Mutational analysis of phospholipase A2A: a positional candidate susceptibility gene for bipolar disorder. Molecular Psychiatry 4 (3), pp.274-279. (10.1038/sj.mp.4000476)
• Jacobsen, N. J. et al., 1999. ATP2A2 mutations in Darier's disease and their relationship to neuropsychiatric phenotypes. Human Molecular Genetics 8 (9), pp.1631-1636. (10.1093/hmg/8.9.1631)
• Jones, I. R. , Owen, M. J. and Craddock, N. J. 1999. Further evidence of familial cosegregation of major affective disorder and Darier's disease. Molecular Psychiatry 4 , pp.S73-S73.
• Jones, L. A. et al., 1999. Sustained and selective attention as quantitative measures of schizophrenia liability. Molecular Psychiatry 4 , pp.S117-S117.
• Jonsson, E. G. et al., 1999. Monoamine oxidase a promoter polymorphism and relationships to monoamine metabolite concentrations in CSF. Molecular Psychiatry 4 , pp.S97-S98.
• Kehoe, P. et al., 1999. Age of onset in Huntington disease: sex specific influence of apolipoprotein E genotype and normal CAG repeat length. Journal of Medical Genetics 36 (2), pp.108-111. (10.1136/jmg.36.2.108)
• Kehoe, P. et al., 1999. A full genome scan for late onset Alzheimer's disease. Human Molecular Genetics 8 (2), pp.237-245. (10.1093/hmg/8.2.237)
• Kehoe, P. G. et al., 1999. Variation in DCP1, encoding ACE, is associated with susceptibility to Alzheimer disease. Nature Genetics 21 (1), pp.71-72. (10.1038/5009)
• Kerwin, R. and Owen, M. J. 1999. Genetics of novel therapeutic targets in schizophrenia. The British journal of psychiatry. Supplement. 38 , pp.1-4.
• Kirov, G. et al. 1999. Family-based association studies of bipolar disorder with candidate genes involved in dopamine neurotransmission: DBH, DAT1, COMT, DRD2, DRD3 and DRD5. Molecular Psychiatry 4 (6), pp.558-565.
• Kirov, G. et al. 1999. A functional polymorphism in the promoter of monoamine oxidase A gene and bipolar affective disorder. International Journal of Neuropsychopharmacology 2 (4), pp.293-298. (10.1017/s1461145799001601)
• Kirov, G. and Owen, M. J. 1999. Identification of new microsatellite markers on chromosome 4p16. American Journal of Human Genetics 65 (4), pp.A257-A257.
• Kirov, G. et al. 1999. Tryptophan hydroxylase gene and manic-depressive illness [letter]. Archives of General Psychiatry 56 (1), pp.98-99. (10.1001/archpsyc.56.1.98)
• Kirov, G. et al. 1999. Bipolar disorder and the serotonin transporter gene: a family-based association study. Psychological Medicine -London- 29 (5), pp.1249-1254. (10.1017/s003329179900882x)
• McCarthy, G. et al., 1999. Familial aggregation of clinical variables in a sibling pair study of schizophrenia. Molecular Psychiatry 4 , pp.S121-S121.
• McCarthy, G. et al., 1999. Intra-pair correlations of symptom dimensions in a sibling pair study of schizophrenia. Molecular Psychiatry 4 , pp.S26-S27.
• Middle, F. A. et al., 1999. Association studies between bipolar disorder and a polymorphism in the Wolfram gene. Molecular Psychiatry 4 , pp.S76-S76.
• Murphy, K. et al., 1999. The prevalence of chromosome 22q11 deletions in an adult congenital heart disease population. Journal of Medical Genetics 36 , pp.S59-S59.
• Murphy, K. C. , Jones, L. A. and Owen, M. J. 1999. High rates of schizophrenia in adults with velo-cardio-facial syndrome. Archives of General Psychiatry 56 (10), pp.940-945. (10.1001/archpsyc.56.10.940)
• Murphy, K. C. , Jones, L. A. and Owen, M. J. 1999. High rates of schizophrenia in velo-cardio-facial syndrome. American Journal of Human Genetics 65 (4), pp.A336-A336.
• Murphy, K. C. and Owen, M. J. 1999. No association between polymorphisms of catechol-O-methyltransferase (COMT) or monoamine oxidase (MAO) genes and schizophrenia in adults with velo-cardio-facial syndrome. Journal of Medical Genetics 36 , pp.S78-S78.
• Norton, N. et al., 1999. No evidence for association between schizophrenia and MAO-A promoter polymorphism. Molecular Psychiatry 4 , pp.S96-S96.
• O'Donovan, M. C. and Owen, M. J. 1999. Candidate-gene association studies of schizophrenia. American Journal of Human Genetics 65 (3), pp.587-592. (10.1086/302560)
• Owen, M. J. 1999. Evidence of novel loci in late onset Alzheimer's disease. Molecular Psychiatry 4 (1), pp.S9-S9.
• Owen, M. J. and Cardno, A. G. 1999. Psychiatric genetics: progress, problems, and potential. The Lancet 354 (1), pp.SI11-SI14. (10.1016/S0140-6736(99)90242-8)
• Rees, M. I. et al., 1999. Autosome search for schizophrenia susceptibility genes in multiply affected families. Molecular Psychiatry 4 (4), pp.353-359. (10.1038/sj.mp.4000521)
• Sakuntabhai, A. et al., 1999. Mutations in ATP2A2, encoding a Ca2+ pump, cause Darier disease. Nature Genetics 21 (3), pp.271-277.
• Sanders, R. D. et al., 1999. Pre-pulse inhibition in the functional psychoses. Molecular Psychiatry 4 , pp.S121-S121.
• Thapar, A. et al. 1999. A quantitative trait loci study of attention deficit hyperactivity disorder. Molecular Psychiatry 4 , pp.S52-S53.
• Wavrant-De Vrieze, F. et al., 1999. Genetic variability at the amyloid-beta precursor protein locus may contribute to the risk of late-onset Alzheimer's disease. Neuroscience Letters 269 (2), pp.67-70.
• Wavrant-DeVrieze, F. et al., 1999. No association between the alpha-2 macroglobulin I1000V polymorphism and Alzheimer's disease. Neuroscience Letters 262 (2), pp.137-139. (10.1016/S0304-3940(99)00035-X)
• Williams, H. J. et al., 1999. Characterization, mutational analysis, and association of the human proline oxidase gene: A candidate gene for schizophrenia.. Molecular Psychiatry 4 , pp.S115-S115.
• Williams, J. , Kehoe, P. G. and Owen, M. J. 1999. Case-control study of presenelin-1 intronic polymorphism. Journal of Neurology, Neurosurgery & Psychiatry 66 (6), pp.702. (10.1136/jnnp.66.6.702)
• Williams, N. M. et al. 1999. Polymorphism screening and association studies of the Wolfram syndrome gene in schizophrenia. Molecular Psychiatry 4 (supp1), pp.S42-S42.
• Williams, N. M. et al. 1999. A two-stage genome scan for schizophrenia susceptibility genes in 196 affected sibling pairs. Human Molecular Genetics 8 (9), pp.1729-1739. (10.1093/hmg/8.9.1729)
• Willliams, H. et al., 1999. No evidence for allelic association between schizophrenia and a functional variant of the human dopamine beta-hydroxylase gene (DBH).. American Journal Of Medical Genetics Part A 88 (5), pp.557-559. (10.1002/(SICI)1096-8628(19991015)88:5<557::AID-AJMG22>3.0.CO;2-F)

1998

• Arranz, M. J. et al., 1998. Evidence for association between polymorphisms in the promoter and coding regions of the 5-HT2A receptor gene and response to clozapine. Molecular Psychiatry 3 (1), pp.61-66. (10.1038/sj.mp.4000348)
• Asherson, P. et al., 1998. A study of chromosome 4p markers and dopamine D5 receptor gene in schizophrenia and bipolar disorder. Molecular Psychiatry 3 (4), pp.310-320. (10.1038/sj.mp.4000399)
• Ball, D. et al., 1998. Dopamine markers and general cognitive ability. NeuroReport 9 (2), pp.347-349. (10.1097/00001756-199801260-00031)
• Bennett, P. et al., 1998. The Wellcome Trust UK-Irish Bipolar Sib-pair Study: Chromosome 21. American Journal of Medical Genetics 81 (6), pp.541-541.
• Bowen, T. et al. 1998. Further support for an association between a polymorphic CAG repeat in the hKCa3 gene and schizophrenia. Molecular Psychiatry 3 (3), pp.266-269. (10.1038/sj.mp.4000400)
• Bowen, T. et al. 1998. Linked polymorphisms upstream of exons 1 and 2 of the human cholecystokinin gene are not associated with schizophrenia or bipolar disorder. Molecular Psychiatry 3 (1), pp.67-71. (10.1038/sj.mp.4000293)
• Cardno, A. G. et al., 1998. Sibling pairs with schizophrenia or schizoaffective disorder: associations of subtypes, symptoms and demographic variables. Psychological Medicine 28 (4), pp.815-823. (10.1017/S0033291798006783)
• Cardno, A. G. et al., 1998. Association between functional psychosis and expanded CAG/CTG repeats is not explained by health stratification [comparative study]. Psychiatric Genetics 8 (1), pp.29-32. (10.1097/00041444-199800810-00005)
• Cardno, A. G. et al., 1998. Polydactyly and psychosis. Five cases of co-occurrence. British Journal of Psychiatry 172 , pp.184-185. (10.1192/bjp.172.2.184)
• Chorney, M. et al., 1998. A Quantitative Trait Locus Associated With Cognitive Ability in Children. Psychological Science 9 (3), pp.159-166. (10.1111/1467-9280.00032)
• Daniels, J. et al., 1998. A simple method for analyzing microsatellite allele image patterns generated from DNA pools and its application to allelic association studies. American Journal of Human Genetics 62 (5), pp.1189-1197. (10.1086/301816)
• Daniels, J. et al., 1998. Molecular genetic studies of cognitive ability. Human Biology 70 (2), pp.281-296.
• Deb, S. et al., 1998. No significant association between a PS-1 intronic polymorphism and dementia in Down's syndrome. Alzheimers Reports 1 (6), pp.365-368.
• Fisher, P. J. et al., 1998. DNA pooling for genomic scanning - Application to an association study. American Journal of Medical Genetics 81 (6), pp.469-469.
• Hill, L. et al., 1998. QTLs for general cognitive ability in children: DNA pooling for chromosome 22. American Journal of Medical Genetics 81 (6), pp.486-486.
• Kehoe, P. G. et al., 1998. The butyrylcholinesterase K variant and susceptibility to Alzheimer's disease. Journal of Medical Genetics 35 (12), pp.1034-1035. (10.1136/jmg.35.12.1034)
• Kirov, G. et al. 1998. Family-based association studies of candidate genes in bipolar disorder. American Journal of Medical Genetics 81 (6), pp.476-477.
• Kirov, G. et al. 1998. Low activity allele of catechol-O-methyltransferase gene associated with rapid cycling bipolar disorder. Molecular Psychiatry 3 (4), pp.342-345. (10.1038/sj.mp.4000385)
• Murphy, K. C. et al., 1998. Chromosome 22q11 deletions - An under-recognized cause of idiopathic learning disability?. Schizophrenia Research 29 (1-2), pp.132-132. (10.1016/S0920-9964(97)88634-3)
• Murphy, K. C. et al., 1998. Chromosome 22qII deletions. An under-recognised cause of idiopathic learning disability. British Journal of Psychiatry 172 , pp.180-183. (10.1192/bjp.172.2.180)
• Murphy, K. C. and Owen, M. J. 1998. Chromosome 22qII deletions and aggressive behaviour. British Journal of Psychiatry 173 , pp.353-354. (10.1192/bjp.173.4.353b)
• Murphy, K. C. and Owen, M. J. 1998. The behavioural phenotype in velo-cardio-facial syndrome [Conference Abstract]. Schizophrenia Research 29 (1-2), pp.132-132. (10.1016/S0920-9964(97)88633-1)
• Murphy, K. C. et al., 1998. A linkage study of chromosome 22q in SIB-pairs with schizophrenia. Schizophrenia Research 29 (1-2), pp.131-132. (10.1016/S0920-9964(97)88632-X)
• Owen, M. J. 1998. Psychiatric disorders in Wolfram syndrome heterozygotes. Molecular Psychiatry 3 (1), pp.12-13.
• Owen, M. J. and Craddock, N. J. 1998. Chromosome 11 workshop. Psychiatric Genetics 8 (2), pp.89-92.
• Owen, M. J. and McGuffin, P. 1998. The programmed alliance of two disciplines. La Recherche 311 , pp.38-39.
• Poulton, K. et al., 1998. A molecular genetic study of hyperkinetic disorder/attention deficit hyperactivity disorder. American Journal of Medical Genetics 81 (6), pp.458-458.
• Spurlock, G. et al. 1998. A family based association study of T102C polymorphism in 5HT2A and schizophrenia plus identification of new polymorphisms in the promoter. Molecular Psychiatry 3 (1), pp.42-49. (10.1038/sj.mp.4000342)
• Spurlock, G. et al., 1998. European Multicentre Association Study of Schizophrenia: a study of the DRD2 Ser311Cys and DRD3 Ser9Gly polymorphisms. American Journal of Medical Genetics 81 (1), pp.24-28. (10.1002/(SICI)1096-8628(19980207)81:1<24::AID-AJMG5>3.0.CO;2-N)
• Spurlock, G. et al. 1998. Polymorphism screening of the human type 1 sigma (sigma) receptor (a candidate gene for schizophrenia) using a modified dideoxy fingerprinting technique.. American Journal of Medical Genetics 81 (6), pp.525-526.
• Straub, R. E. et al., 1998. A transmission disequilibrium and linkage analysis of D22S278 marker alleles in 574 families: further support for a susceptibility locus for schizophrenia at 22q12. Schizophrenia Collaborative Linkage Group for Chromosome 22. Schizophrenia Research 32 (2), pp.115-121. (10.1016/S0920-9964(98)00048-6)
• Vallada, H. P. et al., 1998. Linkage analysis between bipolar affective disorder and markers on chromosome X. Psychiatric Genetics 8 (3), pp.183-186. (10.1097/00041444-199800830-00008)
• Williams, J. et al. 1998. A meta-analysis and transmission disequilibrium study of association between the dopamine D3 receptor gene and schizophrenia. Molecular Psychiatry 3 (2), pp.141-149.
• Williams, N. M. , Fenton, I. and Owen, M. J. 1998. PsycheMap - An interactive Internet-based database of psychiatric genetics linkage, association, and genome mapping projects. American Journal of Medical Genetics 81 (6), pp.463-464.
• Williams, N. M. et al. 1998. Screening the critical region on chromosome 4p with DNA pooling for association with bipolar disorder. American Journal of Medical Genetics 81 (6), pp.486-486.
• Wright, P. et al., 1998. A transmission/disequilibrium study of the DRB1*04 gene locus on chromosome 6p21.3 with schizophrenia. Schizophrenia Research 32 (2), pp.75-80. (10.1016/S0920-9964(98)00050-4)
• Wu, W. S. et al., 1998. Genetic studies on chromosome 12 in late-onset Alzheimer disease. JAMA - The Journal of the American Medical Association 280 (7), pp.619-622. (10.1001/jama.280.7.619)

1997

• Andrew, M. and Owen, M. J. 1997. Hyperekplexia: abnormal startle response due to glycine receptor mutations. British Journal of Psychiatry 170 , pp.106-108. (10.1192/bjp.170.2.106)
• Arranz, M. et al., 1997. Polymorphisms in the 5-HT2A receptor gene and promoter region associated with clozapine response. Schizophrenia Research 24 (1-2), pp.90. (10.1016/S0920-9964(97)82243-8)
• Craddock, N. J. et al. 1997. Expanded CAG/CTG repeats in bipolar disorder: no correlation with phenotypic measures of illness severity. Biological psychiatry 42 (10), pp.876-881. (10.1016/s0006-3223(96)00516-1)
• Daniels, J. K. et al., 1997. Linkage study of chromosome 6p in sib-pairs with schizophrenia. American Journal of Medical Genetics 74 (3), pp.319-323. (10.1002/(SICI)1096-8628(19970531)74:3<319::AID-AJMG14>3.0.CO;2-R)
• Davies, S. J. et al., 1997. Parietal foramina and multiple exostoses - familial inheritance of a molecular deletion. American Journal of Human Genetics 61 (4), pp.A95-A95.
• Guy, C. et al. 1997. Exclusion of expansion of 50 CAG/CTG trinucleotide repeats in bipolar disorder. American Journal of Psychiatry 154 (8), pp.1146-1147.
• Jones, L. et al. 1997. No evidence for expanded polyglutamine sequences in bipolar disorder and schizophrenia. Molecular Psychiatry 2 (6), pp.478-482. (10.1038/sj.mp.4000297)
• Jonsson, E. G. et al., 1997. Tryptophan hydroxylase and catechol-O-methyltransferase gene polymorphisms: relationships to monoamine metabolite concentrations in CSF of healthy volunteers. European Archives of Psychiatry and Clinical Neuroscience 247 (6), pp.297-230. (10.1007/BF02922258)
• Kunugi, H. et al., 1997. Catechol-O-methyltransferase polymorphisms and schizophrenia: a transmission disequilibrium study in multiply affected families. Psychiatric Genetics 7 (3), pp.97-101. (10.1097/00041444-199723000-00001)
• Murphy, K. C. et al., 1997. Chromosome 22q11 deletions: An underrecognized cause of idiopathic learning disability?. American Journal of Medical Genetics 74 (6), pp.567-567.
• Murphy, K. C. and Owen, M. J. 1997. The behavioral phenotype in velo-cardio-facial syndrome [Poster Abstract]. American Journal of Medical Genetics 74 (6), pp.660-660. (10.1002/(SICI)1096-8628(19971121)74:6%3C641::AID-AJMG6%3E3.0.CO;2-M)
• Owen, M. J. , Holmans, P. A. and McGuffin, P. 1997. Association studies in psychiatric genetics. Molecular Psychiatry 2 (4), pp.270-273. (10.1038/sj.mp.4000292)
• Owen, M. J. and McGuffin, P. 1997. Genetics and psychiatry. British Journal of Psychiatry 171 , pp.201-202. (10.1192/bjp.171.3.201)
• Petrill, S. A. et al., 1997. No association between general cognitive ability and the A1 allele of the D2 dopamine receptor gene. Behavior Genetics 27 (1), pp.29-31. (10.1023/A:1025659124405)
• Petrill, S. A. et al., 1997. Failure to replicate a QTL association between a DNA marker identified by EST00083 and IQ. Intelligence 25 (3), pp.179-184. (10.1016/S0160-2896(97)90041-6)
• Rees, M. et al., 1997. Association studies of bipolar disorder at the human serotonin transporter gene (hSERT; 5HTT). Molecular Psychiatry 2 (5), pp.398-402. (10.1038/sj.mp.4000256)
• Speight, G. et al., 1997. Exclusion of CAG/CTG trinucleotide repeat loci which map to chromosome 4 in bipolar disorder and schizophrenia. American Journal of Medical Genetics 74 (2), pp.204-206. (10.1002/(SICI)1096-8628(19970418)74:2<204::AID-AJMG19>3.0.CO;2-M)
• Spurlock, G. et al., 1997. Meta-analysis of association studies between schizophrenia and polymorphisms of the 5-hydroxytryptamine type 2A receptor gene. Schizophrenia Research 24 (1-2), pp.91. (10.1016/S0920-9964(97)82247-5)
• Thomas, N. S. T. et al. 1997. Molecular genetic studies in familial Rett syndrome. European Child & Adolescent Psychiatry 6 (Supp 1), pp.94-94.
• Vallada, H. P. et al., 1997. Linkage studies in bipolar affective disorder with markers on chromosome Xq25-27. American Journal of Medical Genetics 74 (6), pp.679-680.
• Williams, J. et al. 1997. A meta analysis of association between the 5-HT2a receptor, T102C polymorphism and schizophrenia. American Journal of Medical Genetics 74 (6), pp.612-612.
• Williams, J. et al. 1997. Meta-analysis of association between the 5-HT2a receptor T102C polymorphism and schizophrenia.. The Lancet 349 (9060), pp.1221. (10.1016/S0140-6736(05)62413-0)
• Williams, N. M. et al. 1997. Association between schizophrenia and a microsatellite polymorphism at the dopamine D5 receptor gene. Psychiatric Genetics 7 (2), pp.83-85. (10.1097/00041444-199722000-00005)
• Williams, N. M. et al. 1997. No evidence for an allelic association between schizophrenia and markers D22S278 and D22S283. American Journal of Medical Genetics 74 (1), pp.37-39. (10.1002/(SICI)1096-8628(19970221)74:1<37::AID-AJMG8>3.0.CO;2-S)

1996

• Asherson, P. et al., 1996. Linkage, association and mutational analysis of the dopamine D3 receptor gene in schizophrenia. Molecular Psychiatry 1 (2), pp.125-132.
• Ashworth, A. et al., 1996. Linkage analysis of the fragile X gene FMR-1 and schizophrenia: no evidence for linkage but report of a family with schizophrenia and an unstable triplet repeat. Psychiatric Genetics 6 (2), pp.81-86. (10.1097/00041444-199622000-00008)
• Bowen, T. et al. 1996. Expansion of 50 CAG/CTG repeats excluded in schizophrenia by application of a highly efficient approach using repeat expansion detection and a PCR screening set. American Journal of Human Genetics 59 (4), pp.912-917.
• Cardno, A. G. et al., 1996. Factor analysis of schizophrenic symptoms using the OPCRIT checklist. Schizophrenia Research 22 (3), pp.233-239. (10.1016/S0920-9964(96)00060-6)
• Cardno, A. G. et al., 1996. Expanded CAG/CTG repeats in schizophrenia. A study of clinical correlates. British Journal of Psychiatry 169 (6), pp.766-771. (10.1192/bjp.169.6.766)
• Cardno, A. et al., 1996. Factor-derived subsyndromes of schizophrenia and familial morbid risks. Schizophrenia Research 18 (2-3), pp.IVA2-IVA2.
• Craddock, N. J. et al. 1996. Concurrent validity of the OPCRIT diagnostic system. Comparison of OPCRIT diagnoses with consensus best-estimate lifetime diagnoses. British Journal of Psychiatry 169 (1), pp.58-63. (10.1192/bjp.169.1.58)
• Craddock, N. J. et al. 1996. Increasing the efficiency of genomic searches for linkage in complex disorders by DNA pooling of affected sib-pairs. Molecular Psychiatry 1 (1), pp.59-64.
• Craddock, N. J. , O'Donovan, M. C. and Owen, M. J. 1996. Introducing Selfcite 2.0--career enhancing software. BMJ 313 (7072), pp.1659-1660. (10.1136/bmj.313.7072.1659)
• Craddock, N. J. and Owen, M. J. 1996. Candidate gene association studies in psychiatric genetics: a SERTain future?. Molecular Psychiatry 1 (6), pp.434-436.
• Craddock, N. J. and Owen, M. J. 1996. Modern molecular genetic approaches to psychiatric disease. British Medical Bulletin 52 (3), pp.434-452. (10.1093/oxfordjournals.bmb.a011558)
• Daniels, J. , McGuffin, P. and Owen, M. J. 1996. Molecular genetic research on IQ: can it be done? Should it be done?. Journal of Biosocial Science 28 (4), pp.491-507. (10.1017/S0021932000022550)
• Daniels, J. K. et al., 1996. No evidence for allelic association between schizophrenia and a polymorphism determining high or low catechol O-methyltransferase activity. American Journal of Psychiatry 153 (2), pp.268-270. (10.1176/ajp.153.2.268)
• Farmer, A. and Owen, M. J. 1996. Genomics: the next psychiatric revolution?. British Journal of Psychiatry 169 (2), pp.135-138. (10.1192/bjp.169.2.135)
• Gill, M. et al., 1996. A combined analysis of D22S278 marker alleles in affected sib-pairs: support for a susceptibility locus for schizophrenia at chromosome 22q12. Schizophrenia Collaborative Linkage Group (Chromosome 22).. American Journal of Medical Genetics 67 (1), pp.40-45. (10.1002/(SICI)1096-8628(19960216)67:1<40::AID-AJMG6>3.0.CO;2-W)
• Jacobsen, N. et al., 1996. Association study of bipolar disorder at the phospholipase A2 gene (PLA2A) in the Darier's disease (DAR) region of chromosome 12q23-q24.1. Psychiatric Genetics 6 (4), pp.195-199. (10.1097/00041444-199624000-00005)
• Kehoe, P. et al., 1996. Association between a PS-1 intronic polymorphism and late onset Alzheimer's disease. NeuroReport 7 (13), pp.2155-2158. (10.1097/00001756-199609020-00019)
• Kehoe, P. et al., 1996. Presenilin-1 polymorphism and Alzheimer's disease. The UK Alzheimer's Disease Collaborative Group. The Lancet 347 (9009), pp.1185.
• Kunugi, H. et al., 1996. A linkage study of schizophrenia with DNA markers from chromosome 8p21-p22 in 25 multiplex families. Schizophrenia Research 22 (1), pp.61-68. (10.1016/0920-9964(96)00048-5)
• Levinson, D. F. et al., 1996. Additional support for schizophrenia linkage on chromosomes 6 and 8: a multicenter study. Schizophrenia Linkage Collaborative Group for Chromosomes 3, 6 and 8. American Journal of Medical Genetics 67 (6), pp.580-594. (10.1002/(SICI)1096-8628(19961122)67:6<580::AID-AJMG11>3.0.CO;2-P)
• Lim, L. C. C. et al., 1996. Assessing the statistical power to detect linkage in a sample of 51 bipolar affective disorder pedigrees. Behavior Genetics 26 (2), pp.113-122. (10.1007/bf02359889)
• Lin, M. W. et al., 1996. Suggestive evidence for linkage of schizophrenia to markers on chromosome 13q14.1-q32. Cytogenetics and Cell Genetics 75 , pp.2-3.
• Murphy, K. C. and Owen, M. J. 1996. Minor physical anomalies and their relationship to the aetiology of schizophrenia. British Journal of Psychiatry 168 (2), pp.139-142. (10.1192/bjp.168.2.139)
• Murphy, K. C. and Owen, M. J. 1996. Schizophrenia, CATCH 22 and FISH. British Journal of Psychiatry 168 (4), pp.397-398.
• O'Donovan, M. C. et al. 1996. Involvement of expanded trinucleotide repeats in common diseases. The Lancet 348 (9043), pp.1739-1740. (10.1016/s0140-6736(05)65870-9)
• O'Donovan, M. C. et al. 1996. Confirmation of association between expanded CAG/CTG repeats and both schizophrenia and bipolar disorder. Psychological Medicine -London- 26 (6), pp.1145-1153. (10.1017/s0033291700035868)
• O'Donovan, M. C. and Owen, M. J. 1996. Dynamic mutations and psychiatric genetics. Psychological Medicine 26 (1), pp.1-6. (10.1017/S0033291700033663)
• Owen, M. J. and Craddock, N. J. 1996. Modern molecular genetic approaches to complex traits: implications for psychiatric disorders [review]. Molecular Psychiatry 1 (1), pp.21-26.
• Owen, M. J. , Kehoe, P. and Williams, J. 1996. Presenilin-1 polymorphism and Alzheimer's disease. The Lancet 348 (9024), pp.414. (10.1016/S0140-6736(05)65041-6)
• Parfitt, E. et al., 1996. No evidence for linkage between schizophrenia and eight microsatellite markers on chromosome 19. Human Heredity 46 (4), pp.191-196. (10.1159/000154352)
• Petrill, S. A. et al., 1996. DNA markers associated with general and specific cognitive abilities. Intelligence 23 (3), pp.191. (10.1016/S0160-2896(96)90003-3)
• Vallada, H. et al., 1996. Linkage studies in bipolar affective disorder with markers on chromosome 21. Journal of affective disorders 41 (3), pp.217-221. (10.1016/s0165-0327(96)00055-9)
• Williams, J. et al. 1996. Association between schizophrenia and T102C polymorphism of the 5-hydroxytryptamine type 2a-receptor gene. European Multicentre Association Study of Schizophrenia (EMASS) Group. The Lancet 347 (9011), pp.1294-1296. (10.1016/S0140-6736(96)90939-3)

1995

• Arranz, M. et al., 1995. Schizophrenia and the androgen receptor gene: report of a sibship showing co-segregation with Reifenstein syndrome but no evidence for linkage in 23 multiply affected families. American Journal of Medical Genetics 60 (5), pp.377-381. (10.1002/ajmg.1320600506)
• Craddock, N. J. et al. 1995. No evidence for allelic association between bipolar disorder and monoamine oxidase A gene polymorphisms. American Journal of Medical Genetics 60 (4), pp.322-324. (10.1002/ajmg.1320600412)
• Craddock, N. J. et al. 1995. Association study of bipolar disorder using a functional polymorphism (Ser311-->Cys) in the dopamine D2 receptor gene. Psychiatric Genetics 5 (2), pp.63-65.
• Daniels, J. et al., 1995. No association between schizophrenia and polymorphisms within the genes for debrisoquine 4-hydroxylase (CYP2D6) and the dopamine transporter (DAT).. American Journal of Medical Genetics 60 (1), pp.85-87. (10.1002/ajmg.1320600115)
• Dawson, E. et al., 1995. Linkage studies of bipolar disorder in the region of the Darier's disease gene on chromosome 12q23-24.1. American Journal of Medical Genetics 60 (2), pp.94-102. (10.1002/ajmg.1320600203)
• Dawson, E. et al., 1995. Systematic search for major genes in schizophrenia: methodological issues and results from chromosome 12. American Journal of Medical Genetics 60 (5), pp.424-433. (10.1002/ajmg.1320600513)
• Liddell, M. B. , Bayer, A. J. and Owen, M. J. 1995. No evidence that common allelic variation in the Amyloid Precursor Protein (APP) gene confers susceptibility to Alzheimer's disease. Human Molecular Genetics 4 (5), pp.853-858. (10.1093/hmg/4.5.853)
• Lin, M. W. et al., 1995. Suggestive evidence for linkage of schizophrenia to markers on chromosome 13q14.1-q32. Psychiatric Genetics 5 (3), pp.117-126. (10.1097/00041444-199505030-00004)
• McGuffin, P. , Owen, M. J. and Farmer, A. E. 1995. Genetic basis of schizophrenia. The Lancet 346 (8976), pp.678-682. (10.1016/S0140-6736(95)92285-7)
• Mullan, M. et al., 1995. Clinical features of early onset, familial Alzheimer's disease linked to chromosome 14. American Journal of Medical Genetics 60 (1), pp.44-52. (10.1002/ajmg.1320600109)
• O'Donovan, M. C. et al. 1995. Expanded CAG repeats in schizophrenia and bipolar disorder [letter]. Nature Genetics 10 (4), pp.380-381. (10.1038/ng0895-380)
• Okoro, C. et al., 1995. No evidence for linkage between the X-chromosome marker DXS7 and schizophrenia. American Journal of Medical Genetics 60 (5), pp.461-464. (10.1002/ajmg.1320600520)
• Plomin, R. et al., 1995. Allelic associations between 100 DNA markers and high versus low IQ. Intelligence 21 (1), pp.31. (10.1016/0160-2896(95)90037-3)
• Roberts, A. G. et al., 1995. Partial characterization and assignment of the gene for protoporphyrinogen oxidase and variegate porphyria to human chromosome 1q23. Human Molecular Genetics 4 (12), pp.2387-2390. (10.1093/hmg/4.12.2387)
• Skuder, P. et al., 1995. A polymorphism in mitochondrial DNA associated with IQ?. Intelligence 21 (1), pp.1-11. (10.1016/0160-2896(95)90035-7)
• Vallada, H. et al., 1995. Chromosome 22 markers demonstrate transmission disequilibrium with schizophrenia. Psychiatric Genetics 5 (3), pp.127-130. (10.1097/00041444-199505030-00005)
• Vallada, H. P. et al., 1995. Linkage studies on chromosome 22 in familial schizophrenia. American Journal of Medical Genetics 60 (2), pp.139-146. (10.1002/ajmg.1320600210)

1994

• Asherson, P. and Owen, M. J. 1994. Anticipation in mental illness. The American Journal of Human Genetics 54 (2), pp.386-387.
• Asherson, P. et al., 1994. Imprinting and anticipation. Are they relevant to genetic studies of schizophrenia?. The British Journal of Psychiatry 164 (5), pp.619-624. (10.1192/bjp.164.5.619)
• Asherson, P. et al., 1994. DRD2 Ser311/Cys311 polymorphism in schizophrenia. The Lancet 343 (8904), pp.1045-1045.
• Craddock, N. J. et al. 1994. Bipolar affective puerperal psychosis associated with consanguinity. British Journal of Psychiatry 164 (3), pp.359-364. (10.1192/bjp.164.3.359)
• Craddock, N. J. et al. 1994. Variation at the fragile X locus does not influence susceptibility to bipolar disorder. American Journal of Medical Genetics 54 (2), pp.141-143. (10.1002/ajmg.1320540209)
• Craddock, N. J. , McGuffin, P. and Owen, M. J. 1994. Darier's disease cosegregating with affective disorder [letter]. British Journal of Psychiatry 165 (2), pp.272-272. (10.1192/bjp.165.2.272a)
• Craddock, N. J. and Owen, M. J. 1994. Chromosomal aberrations and bipolar affective disorder [review]. British Journal of Psychiatry 164 (4), pp.507-512. (10.1192/bjp.164.4.507)
• Craddock, N. J. and Owen, M. J. 1994. Is there an inverse relationship between Down's syndrome and bipolar affective disorder? literature review and genetic implications [Review]. Journal of Intellectual Disability Research 38 (6), pp.613-620. (10.1111/j.1365-2788.1994.tb00462.x)
• Craddock, N. J. et al. 1994. Familial cosegregation of major affective disorder and Darier's disease (keratosis follicularis). British Journal of Psychiatry 164 (3), pp.355-358. (10.1192/bjp.164.3.355)
• Daniels, J. et al., 1994. Repeat length variation in the dopamine D4 receptor gene shows no evidence of association with schizophrenia. American Journal of Medical Genetics 54 (3), pp.256-258. (10.1002/ajmg.1320540313)
• Daniels, J. et al., 1994. IQ and variation in the number of fragile X CGG repeats: No association in a normal sample. Intelligence 19 (1), pp.45-50. (10.1016/0160-2896(94)90052-3)
• James, C. M. et al., 1994. Debrisoquine hydroxylase gene polymorphism in motor-neuron disease. Neurodegeneration 3 (2), pp.149-152.
• Korner, J. et al., 1994. Association and haplotype analysis at the tyrosine hydroxylase locus in a combined German-British sample of manic depressive patients and controls. Psychiatric Genetics 4 (3), pp.167-175. (10.1097/00041444-199400430-00007)
• Liddell, M. B. et al. 1994. Confirmation of association between the e4 allele of apolipoprotein E and Alzheimer's disease. Journal of Medical Genetics 31 (3), pp.197-200. (10.1136/jmg.31.3.197)
• Mant, R. et al., 1994. Relationship between homozygosity at the dopamine D3 receptor gene and schizophrenia. American Journal of Medical Genetics 54 (1), pp.21-26. (10.1002/ajmg.1320540106)
• McGuffin, P. et al., 1994. The strength of the genetic effect. Is there room for an environmental influence in the aetiology of schizophrenia?. British Journal of Psychiatry 164 (5), pp.593-599. (10.1192/bjp.164.5.593)
• McGuffin, P. et al., 1994. Genetics, chance and dysmorphogenesis in schizophrenia. British Journal of Psychiatry 165 , pp.694-695. (10.1192/bjp.165.5.693b)
• McGuffin, P. et al., 1994. Seminars in psychiatric genetics. Gaskell, Royal College of Psychiatrists.
• Owen, M. J. 1994. The molecular genetics of Alzheimer's disease. Molecular and Cell Biology of Human Diseases Series 1994 (4), pp.92-109.
• Owen, M. J. , Liddell, M. and McGuffin, P. 1994. Alzheimer's disease. British Medical Journal (BMJ) 308 (6930), pp.672-673. (10.1136/bmj.308.6930.672)
• Parfitt, E. et al., 1994. The gene for Darier's disease maps between D12S78 and D12S79. Human Molecular Genetics 3 (1), pp.35-38. (10.1093/hmg/3.1.35)
• Plomin, R. , Owen, M. J. and McGuffin, P. 1994. The genetic basis of complex human behaviors. Science 264 (5166), pp.1733-1739. (10.1126/science.8209254)
• Plomin, R. et al., 1994. DNA markers associated with high versus low IQ: The IQ quantitative trait loci (QTL) project. Behavior Genetics 24 (2), pp.107-118. (10.1007/BF01067815)
• Pulver, A. E. et al., 1994. Follow-up of a report of a potential linkage for schizophrenia on chromosome 22q12-q13.1: Part 2. American Journal of Medical Genetics 54 (1), pp.44-50. (10.1002/ajmg.1320540109)
• Rees, M. I. et al., 1994. Evidence for recessive as well as dominant forms of startle disease (hyperekplexia) caused by mutations in the alpha 1 subunit of the inhibitory glycine receptor. Human Molecular Genetics 3 (12), pp.2175-2179. (10.1093/hmg/3.12.2175)
• Shaikh, S. et al., 1994. Failure to find linkage between a functional polymorphism in the dopamine D4 receptor gene and schizophrenia. American Journal of Medical Genetics 54 (1), pp.8-11. (10.1002/ajmg.1320540104)
• Thapar, A. et al. 1994. The genetics of mental retardation. The British Journal of Psychiatry 164 (6), pp.747. (10.1192/bjp.164.6.747)
• Vallada, H. et al., 1994. Is there a major gene for schizophrenia on chromosome-22. Schizophrenia Research 11 (2), pp.147-147.

1993

• Asherson, P. et al., 1993. Failure to find linkage between schizophrenia and genetic markers on chromosome 21. American Journal of Medical Genetics 48 (3), pp.161-165. (10.1002/ajmg.1320480310)
• Craddock, N. J. et al. 1993. Linkage is excluded between Darier's disease and the Duffy blood group locus in five British families. Annales de Genetique 36 (4), pp.211-213.
• Craddock, N. J. et al. 1993. The gene for Darier's disease maps to chromosome 12q23-q24.1. Human Molecular Genetics 2 (11), pp.1941-1943. (10.1093/hmg/2.11.1941)
• Gill, M. et al., 1993. A linkage study of schizophrenia with DNA markers from the long arm of chromosome 11. Psychological Medicine 23 (1), pp.27-44. (10.1017/S0033291700038824)
• Jones, P. B. et al., 1993. A case-control study of family history and cerebral cortical abnormalities in schizophrenia. Acta Psychiatrica Scandinavica 87 (1), pp.6-12. (10.1111/j.1600-0447.1993.tb03322.x)
• Mant, R. et al., 1993. Susceptibility to schizophrenia and the dopamine d3 receptor gene. American Journal of Human Genetics 53 (3), pp.1654-1654.
• McGuffin, P. and Owen, M. J. 1993. Molecular-genetics and schizophrenia - the current picture. Behavior Genetics 23 (6), pp.558-558.
• Owen, M. J. and McGuffin, P. 1993. Association and linkage: complementary strategies for complex disorders.. Journal of Medical Genetics 30 (8), pp.638-639. (10.1136/jmg.30.8.638)
• Saudino, K. J. et al., 1993. Applying a QTL association approach to temperament. Behavior Genetics 23 (6), pp.564-564.
• Shaikh, S. et al., 1993. The dopamine D3 receptor gene: no association with bipolar affective disorder. Journal of Medical Genetics 30 (4), pp.308-309. (10.1136/jmg.30.4.308)

1992

• Asherson, P. et al., 1992. No evidence for a pseudoautosomal locus for schizophrenia. Linkage analysis of multiply affected families. British Journal of Psychiatry 161 (1), pp.63-68. (10.1192/bjp.161.1.63)
• Collier, D. et al., 1992. Linkage analysis of schizophrenia on chromosome 22. Schizophrenia Research 6 (2), pp.89-90. (10.1016/0920-9964(92)90085-J)
• Craddock, N. J. and Owen, M. J. 1992. Christmas disease and major affective disorder [letter]. British Journal of Psychiatry 160 , pp.715-715. (10.1192/bjp.160.5.715a)
• Dawson, E. et al., 1992. Genetic linkage analysis of schizophrenia using microsatellite repeat polymorphisms on chromosome 12. Schizophrenia Research 6 (2), pp.90-90. (10.1016/0920-9964(92)90086-K)
• Mant, R. et al., 1992. Schizophrenia scepticism. Nature Genetics 2 (1), pp.12-12. (10.1038/ng0992-12)
• Nanko, S. et al., 1992. Linkage Study of Schizophrenia with Markers on Chromosome 11 in Two Japanese Pedigrees. Psychiatry and Clinical Neurosciences 46 (1), pp.155-159. (10.1111/j.1440-1819.1992.tb00828.x)
• O'Donovan, M. C. and Owen, M. J. 1992. Advances and Retreats in the Molecular Genetics of Major Mental Illness. Annals of Medicine 24 (3), pp.171-177. (10.3109/07853899209147816)
• Owen, M. J. and GILL, M. 1992. Looking for genes in schizophrenia. Clinical Neuropharmacology 15 (Suppl1), pp.222A-223A. (10.1097/00002826-199201001-00116)
• Owen, M. J. and Liddell, M. 1992. Role of amyloid beta-protein in Alzheimer's disease. The Lancet 340 (8823), pp.851-851.
• Owen, M. J. et al. 1992. No association between RFLPs at the porphobilinogen deaminase gene and schizophrenia. Human Genetics 90 (1-2), pp.131-132. (10.1007/BF00210756)
• Owen, M. J. and McGuffin, P. 1992. The molecular genetics of schizophrenia.. British Medical Journal (BMJ) 305 (6855), pp.664-665. (10.1136/bmj.305.6855.664)
• Shaikh, S. , Owen, M. J. and Gill, M. 1992. DRD2 (TGn) repeat polymorphism in a large family data set. Schizophrenia Research 6 (2), pp.91-91. (10.1016/0920-9964(92)90088-M)
• Vallada, H. et al., 1992. Debrisoquine 4-hydroxylase (CYP2D) locus and possible susceptibility to schizophrenia. The Lancet 340 (8812), pp.181-182. (10.1016/0140-6736(92)93266-P)

1991

• Foerster, A. et al., 1991. Pre-morbid adjustment and personality in psychosis. Effects of sex and diagnosis. British Journal of Psychiatry 158 (2), pp.171-176. (10.1192/bjp.158.2.171)
• Foerster, A. et al., 1991. Low birth weight and a family history of schizophrenia predict poor premorbid functioning in psychosis. Schizophrenia Research 5 (1), pp.13-20. (10.1016/0920-9964(91)90049-W)
• Goate, A. et al., 1991. Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease. Nature 349 (6311), pp.704-706. (10.1038/349704a0)
• Mant, R. et al., 1991. Exclusion of close linkage between GABA a receptor subunit 1a gene and schizophrenia using a microsatellite repeat marker. Cytogenetics and Cell Genetics 58 (3-4), pp.1900-1991.
• Mant, R. et al., 1991. Mononucleotide repeat polymorphism in the APP gene. Nucleic Acids Research 19 (16), pp.4572-4572. (10.1093/nar/19.16.4572-a)
• McGuffin, P. and Owen, M. J. 1991. The molecular genetics of schizophrenia: An overview and forward view. European Archives of Psychiatry and Clinical Neuroscience 240 (3), pp.169-173. (10.1007/BF02190759)
• Owen, M. J. , Goate, A. and Hardy, J. 1991. A polymorphic microsatellite repeat sequence on chromosome 21 (D21S80). Nucleic Acids Research 19 (16), pp.4574-4571. (10.1093/nar/19.16.4574)
• Owen, M. J. and McGuffin, P. 1991. DNA-and classical genetic markers in schizophrenia. European Archives of Psychiatry and Clinical Neuroscience 240 (3), pp.197-203. (10.1007/BF02190764)
• Owen, M. J. et al. 1991. Linkage studies of schizophrenia with markers of chromosome-11q. American Journal of Human Genetics 49 (4), pp.354-354.
• Upadhyaya, M. et al., 1991. A closely linked DNA marker for facioscapulohumeral disease on chromosome 4q.. Journal of Medical Genetics 28 (10), pp.665-671. (10.1136/jmg.28.10.665)

1990

• Goate, A. et al., 1990. Genetics of Alzheimer's disease. Advances in Neurology -New York- Raven Press- 51 , pp.197-198.
• Owen, M. J. , Craufurd, D. and St Clair, D. 1990. Localisation of a susceptibility locus for schizophrenia on chromosome 5. British Journal of Psychiatry 157 (1), pp.123-127. (10.1192/bjp.157.1.123)
• Owen, M. J. et al. 1990. Physical mapping around the Alzheimer disease locus on the proximal long arm of chromosome 21.. American Journal of Human Genetics 46 (2), pp.316-322.
• Owen, M. J. , McGuffin, P. and Andrews, R. 1990. Obstetric complications and schizophrenia. The Lancet 336 (8707), pp.122-122. (10.1016/0140-6736(90)91643-O)
• Owen, M. J. and Mullan, M. J. 1990. Molecular genetic studies of manic-depression and schizophrenia. Trends in Neurosciences 13 (1), pp.29-31. (10.1016/0166-2236(90)90059-J)
• St George-Hyslop, P. H. et al., 1990. Genetic linkage studies suggest that Alzheimer's disease is not a single homogeneous disorder. Nature 347 (6289), pp.194-197. (10.1038/347194a0)
• Upadhyaya, M. et al., 1990. DNA marker applicable to presymptomatic and prenatal diagnosis of facioscapulohumeral disease. The Lancet 336 (8726), pp.1320-1321. (10.1016/0140-6736(90)93005-A)

1989

• Goate, A. et al., 1989. Predisposing locus for Alzheimer's disease on chromosome 21. The Lancet 333 (8634), pp.352-355. (10.1016/S0140-6736(89)91725-X)
• HARDY, J. A. et al., 1989. Molecular genetics of Alzheimer's disease. Biochemical Society Transactions 17 (1), pp.75-76. (10.1042/bst0170075)
• Hardy, J. et al., 1989. Modelling the occurrence and pathology of Alzheimer's disease. Neurobiology of Aging 10 (5), pp.429-431. (10.1016/0197-4580(89)90084-5)
• Hardy, J. et al., 1989. Presenile dementia associated with mosaic trisomy 21 in a patient with a down syndrome child. The Lancet 334 (8665), pp.743-743. (10.1016/S0140-6736(89)90805-2)
• Owen, M. J. , Lewis, S. W. and Murray, R. M. 1989. Family history and cerebral ventricular enlargement in schizophrenia. A case control study. British Journal of Psychiatry 154 (5), pp.629-634. (10.1192/bjp.154.5.629)

1988

• Owen, M. J. and Lewis, S. W. 1988. Risk factors in schizophrenia. British Journal of Psychiatry 153 (3), pp.407-407. (10.1192/bjp.153.3.407a)
• Owen, M. J. , Lewis, S. W. and Murray, R. M. 1988. Obstetric complications and schizophrenia: a computed tomographic study. Psychological Medicine 18 (02), pp.331-339. (10.1017/S003329170000787X)
• Owen, M. J. and Murray, R. 1988. Blue genes. British Medical Journal (BMJ) 297 (6653), pp.871-872.
• Owen, M. J. and Whatley, S. 1988. Polymorphic DNA markers and mental disease. Psychological Medicine 18 (3), pp.529-533.

1987

• Owen, M. J. and Nimgaonkar, V. 1987. The continuum of psychosis and the gene. British Journal of Psychiatry 150 , pp.566-567.

1986

• Owen, M. J. and Lweis, S. 1986. Lateral ventricular size in schizophrenia. The Lancet 2 (8500), pp.223-224.

1984

• Gaffan, D. et al., 1984. Effects of fornix transection upon associative memory in monkeys: role of the hippocampus in learned action. Quarterly Journal of Experimental Psychology 36 (3), pp.173-221.
• Owen, M. J. and Butler, S. 1984. Does amnesia after transection of the fornix in monkeys reflect abnormal sensitivity to proactive interference?. Behavioural Brain Research 14 (3), pp.183-192.

1981

• Owen, M. J. and Butler, S. 1981. Amnesia after transection of the fornix in monkeys: Long-term memory impaired, short-term memory intact. Behavioural Brain Research 3 (1), pp.115-123. (10.1016/0166-4328(81)90032-2)

1980

• Owen, M. J. and Butler, S. 1980. Effects of fornix lesions on recognition memory in the monkey. Journal of Anatomy 131 (DEC), pp.771-771.
• Owen, M. J. and Butler, S. 1980. The effects of fornix lesions on latent learning in the rat. Physiology & Behavior 24 (5), pp.817-822. (10.1016/0031-9384(80)90133-X)